Laura T. Haas
Affiliations: | Yale University, New Haven, CT |
Area:
Neurodegeneration, Alzheimer's DiseaseGoogle:
"Laura Haas"Mean distance: 15.14 (cluster 32) | S | N | B | C | P |
Parents
Sign in to add mentor| Christian Haass | research assistant | 2012-2012 | Universität München |
| Stephen M. Strittmatter | grad student | 2013- | Yale |
BETA: Related publications
See more...
Publications
|
You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect. |
| Cox TO, Gunther EC, Brody AH, et al. (2019) Anti-PrP antibody rescues cognition and synapses in transgenic alzheimer mice. Annals of Clinical and Translational Neurology. 6: 554-574 |
| Gunther EC, Smith LM, Kostylev MA, et al. (2019) Rescue of Transgenic Alzheimer's Pathophysiology by Polymeric Cellular Prion Protein Antagonists. Cell Reports. 26: 1368 |
| Gunther EC, Smith LM, Kostylev MA, et al. (2019) Rescue of Transgenic Alzheimer's Pathophysiology by Polymeric Cellular Prion Protein Antagonists. Cell Reports. 26: 145-158.e8 |
| Salazar SV, Cox TO, Lee S, et al. (2018) Alzheimer's Disease Risk Factor Pyk2 Mediates Amyloid-β Induced Synaptic Dysfunction and Loss. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience |
| Rammes G, Seeser F, Mattusch K, et al. (2018) The NMDA receptor antagonist Radiprodil reverses the synaptotoxic effects of different amyloid-beta (Aβ) species on long-term potentiation (LTP). Neuropharmacology |
| Salazar SV, Gallardo C, Kaufman AC, et al. (2017) Conditional Deletion of Prnp Rescues Behavioral and Synaptic Deficits after Disease Onset in Transgenic Alzheimer's Disease. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience |
| Haas LT, Salazar SV, Smith LM, et al. (2017) Silent Allosteric Modulation of mGluR5 Maintains Glutamate Signaling while Rescuing Alzheimer's Mouse Phenotypes. Cell Reports. 20: 76-88 |
| Heiss JK, Barrett J, Yu Z, et al. (2016) Early Activation of Experience-Independent Dendritic Spine Turnover in a Mouse Model of Alzheimer's Disease. Cerebral Cortex (New York, N.Y. : 1991) |
| Haas LT, Strittmatter SM. (2016) Oligomers of Amyloid-beta Prevent Physiological Activation of the Cellular Prion Protein-Metabotropic Glutamate Receptor 5 Complex by Glutamate in Alzheimer's Disease. The Journal of Biological Chemistry |
| Haas LT, Salazar SV, Kostylev MA, et al. (2015) Metabotropic glutamate receptor 5 couples cellular prion protein to intracellular signalling in Alzheimer's disease. Brain : a Journal of Neurology |