Estibaliz Capetillo-Zarate
Affiliations: | 2007-2011 | Universidad del Pais Vasco, Leioa, Euskadi, Spain |
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"Estibaliz Capetillo-Zarate"Mean distance: 15.66 (cluster 28) | S | N | B | C | P |
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| Coto-Vilcapoma MA, Castilla-Silgado J, Fernández-García B, et al. (2022) New, Fully Implantable Device for Selective Clearance of CSF-Target Molecules: Proof of Concept in a Murine Model of Alzheimer's Disease. International Journal of Molecular Sciences. 23 |
| Akwa Y, Di Malta C, Zallo F, et al. (2022) Stimulation of synaptic activity promotes TFEB-mediated clearance of pathological MAPT/Tau in cellular and mouse models of tauopathies. Autophagy. 1-18 |
| Rejc L, Gómez-Vallejo V, Joya A, et al. (2022) Longitudinal evaluation of neuroinflammation and oxidative stress in a mouse model of Alzheimer disease using positron emission tomography. Alzheimer's Research & Therapy. 14: 80 |
| Ortiz-Sanz C, Llavero F, Zuazo-Ibarra J, et al. (2022) Recombinant Integrin β1 Signal Peptide Blocks Gliosis Induced by Aβ Oligomers. International Journal of Molecular Sciences. 23 |
| Luchena C, Zuazo-Ibarra J, Valero J, et al. (2022) A Neuron, Microglia, and Astrocyte Triple Co-culture Model to Study Alzheimer's Disease. Frontiers in Aging Neuroscience. 14: 844534 |
| Ortiz-Sanz C, Balantzategi U, Quintela-López T, et al. (2022) Amyloid β / PKC-dependent alterations in NMDA receptor composition are detected in early stages of Alzheimer´s disease. Cell Death & Disease. 13: 253 |
| Rejc L, Gómez-Vallejo V, Joya A, et al. (2021) Longitudinal evaluation of a novel BChE PET tracer as an early biomarker in the brain of a mouse model for Alzheimer disease. Theranostics. 11: 6542-6559 |
| Luchena C, Zuazo-Ibarra J, Alberdi E, et al. (2019) Corrigendum to "Contribution of Neurons and Glial Cells to Complement-Mediated Synapse Removal during Development, Aging and in Alzheimer's Disease". Mediators of Inflammation. 2019: 7539620 |
| Martinsson I, Capetillo-Zarate E, Faideau M, et al. (2019) APP depletion alters selective pre- and post-synaptic proteins. Molecular and Cellular Neurosciences |
| Luchena C, Zuazo-Ibarra J, Alberdi E, et al. (2018) Contribution of Neurons and Glial Cells to Complement-Mediated Synapse Removal during Development, Aging and in Alzheimer's Disease. Mediators of Inflammation. 2018: 2530414 |