Suman Chatterjee, Ph.D.

Affiliations: 
Biological Sciences Duquesne University, Pittsburgh, PA, United States 
Area:
Cell biology
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"Suman Chatterjee"
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Richard Elinson grad student 2013 Duquesne University
 (Differential expression of nodal-signal transducers and global transcriptional repression commit vegetal cells in Eleutherodactylus coqui to form nutritional endoderm.)
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Publications

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Yochum ZA, Cades J, Wang H, et al. (2018) Targeting the EMT transcription factor TWIST1 overcomes resistance to EGFR inhibitors in EGFR-mutant non-small-cell lung cancer. Oncogene
Han J, Goldstein LA, Hou W, et al. (2018) HSP90 inhibition targets autophagy and induces a CASP9-dependent resistance mechanism in NSCLC. Autophagy. 1-14
Yochum ZA, Chatterjee S, Huang EH, et al. (2018) Abstract 5889: TWIST1 is a key mediator of HGF-MET-driven resistance to targeted therapies in EGFR mutant and MET-driven lung cancer Cancer Research. 78: 5889-5889
Chatterjee S, Burns TF. (2017) Targeting Heat Shock Proteins in Cancer: A Promising Therapeutic Approach. International Journal of Molecular Sciences. 18
Yochum ZA, Cades J, Mazzacurati L, et al. (2017) A First-in-Class TWIST1 Inhibitor with Activity in Oncogene-driven Lung Cancer. Molecular Cancer Research : McR
Chatterjee S, Huang EH, Christie I, et al. (2017) Reactivation of the p90RSK-CDC25C pathway leads to bypass of the ganetespib induced G2/M arrest and mediates acquired resistance to ganetespib in KRAS mutant NSCLC. Molecular Cancer Therapeutics
Chatterjee S, Huang EH, Christie I, et al. (2017) Acquired resistance to the Hsp90 inhibitor, ganetespib in KRAS mutant NSCLC is mediated via reactivation of the ERK-p90RSK-mTOR signaling network. Molecular Cancer Therapeutics
Chatterjee S, Huang EH, Christie I, et al. (2017) Abstract 4152: The p90RSK-CDC25C signaling pathway leads to bypass of the ganetespib induced G2/M arrest and mediates acquired resistance to ganetespib inKRASmutant NSCLC Cancer Research. 77: 4152-4152
Chatterjee S, Huang EH, Burns TF. (2016) Abstract 2942: p90 Ribosomal S6 kinase mediates acquired resistance to ganetespib in KRAS mutant NSCLC Cancer Research. 76: 2942-2942
Chatterjee S, Burns TF. (2015) Abstract 762: Ganetespib resistance inKRASmutant NSCLC is mediated through bypassing the G2/M checkpoint and reactivating the PI3K/MTOR pathway Cancer Research. 75: 762-762
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