Andrew D. Kraft, Ph.D.
Affiliations: | University of Wisconsin, Madison, Madison, WI |
Area:
visual working memory, attention, dynamical systems approaches to cognitionGoogle:
"Andrew Kraft"Mean distance: 15.94 (cluster 15) | S | N | B | C | P |
Parents
Sign in to add mentor| Jeffrey S. Johnson | grad student | 2006 | UW Madison | |
| (The role of the Nrf2-antioxidant response element pathway in neuronal support cells during degeneration.) | ||||
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Publications
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| Calkins MJ, Johnson DA, Townsend JA, et al. (2009) The Nrf2/ARE pathway as a potential therapeutic target in neurodegenerative disease. Antioxidants & Redox Signaling. 11: 497-508 |
| Johnson JA, Johnson DA, Kraft AD, et al. (2008) The Nrf2-ARE pathway: an indicator and modulator of oxidative stress in neurodegeneration. Annals of the New York Academy of Sciences. 1147: 61-9 |
| Kraft AD, Resch JM, Johnson DA, et al. (2007) Activation of the Nrf2-ARE pathway in muscle and spinal cord during ALS-like pathology in mice expressing mutant SOD1. Experimental Neurology. 207: 107-17 |
| Jakel RJ, Townsend JA, Kraft AD, et al. (2007) Nrf2-mediated protection against 6-hydroxydopamine. Brain Research. 1144: 192-201 |
| Kraft AD, Lee JM, Johnson DA, et al. (2006) Neuronal sensitivity to kainic acid is dependent on the Nrf2-mediated actions of the antioxidant response element. Journal of Neurochemistry. 98: 1852-65 |
| Lee JM, Li J, Johnson DA, et al. (2005) Nrf2, a multi-organ protector? Faseb Journal : Official Publication of the Federation of American Societies For Experimental Biology. 19: 1061-6 |
| Kraft AD, Johnson DA, Johnson JA. (2004) Nuclear factor E2-related factor 2-dependent antioxidant response element activation by tert-butylhydroquinone and sulforaphane occurring preferentially in astrocytes conditions neurons against oxidative insult. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 24: 1101-12 |
| Shih AY, Johnson DA, Wong G, et al. (2003) Coordinate regulation of glutathione biosynthesis and release by Nrf2-expressing glia potently protects neurons from oxidative stress. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 23: 3394-406 |