Evan C. Ray, Ph.D.
Affiliations: | University of Pennsylvania, Philadelphia, PA, United States |
Area:
Function and assembly of voltage-gated potassium channels.Google:
"Evan Ray"Mean distance: 21.06 (cluster 46)
Parents
Sign in to add mentor| Carol Deutsch | grad student | 2006 | Penn | |
| (The role of intracellular cations in potassium ion channel gating.) | ||||
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Publications
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| Boyd-Shiwarski CR, Weaver CJ, Beacham RT, et al. (2020) Effects of extreme potassium stress on blood pressure and renal tubular sodium transport. American Journal of Physiology. Renal Physiology |
| Aden LA, Pitzer A, Grimes S, et al. (2020) Abstract P131: Jak2 Expression In Cd11c+ Myeloid Cells Plays A Role In Salt-sensitive Hypertension Through An Enac-dependent Mechanism. Hypertension. 76 |
| Pitzer AL, Barbaro NR, Aden L, et al. (2020) Abstract MP38: High Salt Activates The NLRP3 Inflammasome In Antigen Presenting Cells Via ENaC To Promote Salt-Sensitive Hypertension Hypertension. 76 |
| Wang XP, Im SJ, Balchak DM, et al. (2019) Murine epithelial sodium (Na+) channel regulation by biliary factors. The Journal of Biological Chemistry |
| Ray EC, Miller RG, Demko JE, et al. (2018) Urinary Plasmin(ogen) as a Prognostic Factor for Hypertension. Kidney International Reports. 3: 1434-1442 |
| Unruh ML, Pankratz VS, Demko JE, et al. (2017) Trial of Amiloride in Type 2 Diabetes with Proteinuria. Kidney International Reports. 2: 893-904 |
| Ray EC, Chen J, Kelly TN, et al. (2016) Human Epithelial Na+ Channel Missense Variants Identified in the GenSalt Study Alter Channel Activity. American Journal of Physiology. Renal Physiology. ajprenal.00426.2016 |
| Ray EC, Rondon-Berrios H, Boyd CR, et al. (2015) Sodium retention and volume expansion in nephrotic syndrome: implications for hypertension. Advances in Chronic Kidney Disease. 22: 179-84 |
| Ray EC, Kleyman TR. (2015) Cutting it out: ENaC processing in the human nephron. Journal of the American Society of Nephrology : Jasn. 26: 1-3 |
| Ray EC, Deutsch C. (2006) A trapped intracellular cation modulates K+ channel recovery from slow inactivation. The Journal of General Physiology. 128: 203-17 |