michael rowan

Affiliations: 
Trinity College, Hartford, CT, United States 
Area:
synaptic plasticity
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"michael rowan"
Mean distance: 14.77 (cluster 11)
 
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Publications

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Ondrejcak T, Klyubin I, Hu NW, et al. (2023) Tau and Amyloid β Protein in Patient-Derived Aqueous Brain Extracts Act Concomitantly to Disrupt Long-Term Potentiation . The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 43: 5870-5879
Qi Y, Klyubin I, Ondrejcak T, et al. (2021) Enduring glucocorticoid-evoked exacerbation of synaptic plasticity disruption in male rats modelling early Alzheimer's disease amyloidosis. Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology
Babiloni C, Blinowska K, Bonanni L, et al. (2019) What electrophysiology tells us about Alzheimer's disease: a window into the synchronization and connectivity of brain neurons. Neurobiology of Aging. 85: 58-73
Qi Y, Klyubin I, Hu NW, et al. (2019) Pre-plaque Aß-Mediated Impairment of Synaptic Depotentiation in a Transgenic Rat Model of Alzheimer's Disease Amyloidosis. Frontiers in Neuroscience. 13: 861
Ondrejcak T, Hu NW, Qi Y, et al. (2019) Soluble tau aggregates inhibit synaptic long-term depression and amyloid β-facilitated LTD in vivo. Neurobiology of Disease
Ondrejcak T, Klyubin I, Corbett GT, et al. (2018) Cellular Prion Protein Mediates the Disruption of Hippocampal Synaptic Plasticity by Soluble Tau . The Journal of Neuroscience : the Official Journal of the Society For Neuroscience
Hu NW, Corbett GT, Moore S, et al. (2018) Extracellular Forms of Aβ and Tau from iPSC Models of Alzheimer's Disease Disrupt Synaptic Plasticity. Cell Reports. 23: 1932-1938
O'Riordan KJ, Hu NW, Rowan MJ. (2018) Aß Facilitates LTD at Schaffer Collateral Synapses Preferentially in the Left Hippocampus. Cell Reports. 22: 2053-2065
Balschun D, Rowan MJ. (2018) Hippocampal synaptic plasticity in neurodegenerative diseases: Aß, tau and beyond Neuroforum. 24: A133-A141
Zhang D, Qi Y, Klyubin I, et al. (2017) Targeting glutamatergic and cellular prion protein mechanisms of amyloid β-mediated persistent synaptic plasticity disruption: Longitudinal studies. Neuropharmacology
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