Marilee J. Stephens, Ph.D.

Affiliations: 
University of Alberta, Edmonton, Alberta, Canada 
Area:
motor development
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"Marilee Stephens"
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Jaynie F. Yang grad student 2000 University of Alberta
 (The effects of load on normal, infant and Parkinsonian human gait.)
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Publications

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Lin S, Li Y, Lucas-Osma AM, et al. (2019) Locomotor-related V3 interneurons initiate and coordinate muscles spasms after spinal cord injury. Journal of Neurophysiology
Lucas-Osma AM, Li Y, Murray KC, et al. (2019) 5-HT1D receptors inhibit the monosynaptic stretch reflex by modulating C fibre activity. Journal of Neurophysiology
Li Y, Lucas-Osma AM, Black S, et al. (2017) Pericytes impair capillary blood flow and motor function after chronic spinal cord injury. Nature Medicine
Li Y, Li L, Stephens MJ, et al. (2014) Synthesis, transport, and metabolism of serotonin formed from exogenously applied 5-HTP after spinal cord injury in rats. Journal of Neurophysiology. 111: 145-63
Murray KC, Stephens MJ, Rank M, et al. (2011) Polysynaptic excitatory postsynaptic potentials that trigger spasms after spinal cord injury in rats are inhibited by 5-HT1B and 5-HT1F receptors. Journal of Neurophysiology. 106: 925-43
Rank MM, Murray KC, Stephens MJ, et al. (2011) Adrenergic receptors modulate motoneuron excitability, sensory synaptic transmission and muscle spasms after chronic spinal cord injury. Journal of Neurophysiology. 105: 410-22
Murray KC, Stephens MJ, Ballou EW, et al. (2011) Motoneuron excitability and muscle spasms are regulated by 5-HT2B and 5-HT2C receptor activity. Journal of Neurophysiology. 105: 731-48
Murray KC, Nakae A, Stephens MJ, et al. (2010) Recovery of motoneuron and locomotor function after spinal cord injury depends on constitutive activity in 5-HT2C receptors. Nature Medicine. 16: 694-700
Misiaszek JE, Stephens MJ, Yang JF, et al. (2000) Early corrective reactions of the leg to perturbations at the torso during walking in humans. Experimental Brain Research. 131: 511-23
Stephens MJ, Yang JF. (1999) Loading during the stance phase of walking in humans increases the extensor EMG amplitude but does not change the duration of the step cycle. Experimental Brain Research. 124: 363-70
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