Hayley S. McLoughlin, Ph.D.
Affiliations: | 2013 | Neuroscience | University of Iowa, Iowa City, IA |
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Parents
Sign in to add mentor| Beverly L. Davidson | grad student | 2013 | University of Iowa | |
| (MicroRNA regulation of central nervous system development and their species-specific role in evolution.) | ||||
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| Ilnytska O, Lai K, Gorshkov K, et al. (2021) Enrichment of NPC1-deficient cells with the lipid LBPA stimulates autophagy, improves lysosomal function, and reduces cholesterol storage. The Journal of Biological Chemistry. 100813 |
| Costa MDC, Radzwion M, McLoughlin HS, et al. (2020) In Vivo Molecular Signatures of Cerebellar Pathology in Spinocerebellar Ataxia Type 3. Movement Disorders : Official Journal of the Movement Disorder Society |
| Schultz ML, Fawaz MV, Azaria RD, et al. (2019) Synthetic high-density lipoprotein nanoparticles for the treatment of Niemann-Pick diseases. Bmc Medicine. 17: 200 |
| McLoughlin HS, Moore LR, Paulson HL. (2019) Pathogenesis of SCA3 and implications for other polyglutamine diseases. Neurobiology of Disease. 134: 104635 |
| Moore LR, Keller L, Bushart DD, et al. (2019) Antisense oligonucleotide therapy rescues aggresome formation in a novel spinocerebellar ataxia type 3 human embryonic stem cell line. Stem Cell Research. 39: 101504 |
| Zeng L, Zhang D, McLoughlin HS, et al. (2018) Loss of the Spinocerebellar Ataxia type 3 disease protein ATXN3 alters transcription of multiple signal transduction pathways. Plos One. 13: e0204438 |
| McLoughlin HS, Moore LR, Chopra R, et al. (2018) Oligonucleotide therapy mitigates disease in Spinocerebellar Ataxia Type 3 mice. Annals of Neurology |
| Moore LR, Rajpal G, Dillingham IT, et al. (2017) Evaluation of Antisense Oligonucleotides Targeting ATXN3 in SCA3 Mouse Models. Molecular Therapy. Nucleic Acids. 7: 200-210 |
| McLoughlin HS, Wan J, Spengler RM, et al. (2014) Human-specific microRNA regulation of FOXO1: implications for microRNA recognition element evolution. Human Molecular Genetics. 23: 2593-603 |
| McLoughlin HS, Fineberg SK, Ghosh LL, et al. (2012) Dicer is required for proliferation, viability, migration and differentiation in corticoneurogenesis. Neuroscience. 223: 285-95 |