Zi-jun Wang
Affiliations: | Pharmacology and Toxicology | State University of New York, Buffalo, Buffalo, NY, United States |
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"Zi-jun Wang"Mean distance: 16.04 (cluster 19) | S | N | B | C | P |
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Publications
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Wang ZJ, Shwani T, Liu J, et al. (2022) Molecular and cellular mechanisms for differential effects of chronic social isolation stress in males and females. Molecular Psychiatry |
Rapanelli M, Williams JB, Ma K, et al. (2022) Targeting histone demethylase LSD1 for treatment of deficits in autism mouse models. Molecular Psychiatry |
Wang ZJ, Rein B, Zhong P, et al. (2021) Autism risk gene KMT5B deficiency in prefrontal cortex induces synaptic dysfunction and social deficits via alterations of DNA repair and gene transcription. Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology |
Zhang F, Rein B, Zhong P, et al. (2021) Synergistic inhibition of histone modifiers produces therapeutic effects in adult Shank3-deficient mice. Translational Psychiatry. 11: 99 |
Cao Q, Wang W, Williams JB, et al. (2020) Targeting histone K4 trimethylation for treatment of cognitive and synaptic deficits in mouse models of Alzheimer's disease. Science Advances. 6 |
Wei J, Cheng J, Waddell NJ, et al. (2020) DNA Methyltransferase 3A Is Involved in the Sustained Effects of Chronic Stress on Synaptic Functions and Behaviors. Cerebral Cortex (New York, N.Y. : 1991) |
Werner CT, Mitra S, Auerbach BD, et al. (2020) Neuroadaptations in the dorsal hippocampus underlie cocaine seeking during prolonged abstinence. Proceedings of the National Academy of Sciences of the United States of America |
Lepack AE, Werner CT, Stewart AF, et al. (2020) Dopaminylation of histone H3 in ventral tegmental area regulates cocaine seeking. Science (New York, N.Y.). 368: 197-201 |
Rein B, Yan Z, Wang ZJ. (2019) Diminished Social Interaction Incentive Contributes to Social Deficits in Mouse Models of Autism Spectrum Disorder. Genes, Brain, and Behavior. e12610 |
Wang ZJ, Zhong P, Ma K, et al. (2019) Correction: Amelioration of autism-like social deficits by targeting histone methyltransferases EHMT1/2 in Shank3-deficient mice. Molecular Psychiatry |