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Micah J. Maxwell, Ph.D.

Affiliations: 
2012 Johns Hopkins University, Baltimore, MD 
Area:
Neurotransmitters
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"Micah Maxwell"
Mean distance: 14.4 (cluster 40)
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Solomon H. Snyder grad student 2012 Johns Hopkins
 (Inositol polyphosphate multikinase is a physiologic PI3-kinase that activates Akt/PKB.)

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Publications

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Maxwell MJ, Arnold A, Sweeney H, et al. (2021) Unbiased proteomic and phosphoproteomic analysis identifies response signatures and novel susceptibilities after combined MEK and mTOR inhibition in BRAF mutant glioma. Molecular & Cellular Proteomics : McP. 100123
Slavova-Azmanova NS, Kucera N, Satiaputra J, et al. (2013) Gain-of-function Lyn induces anemia: appropriate Lyn activity is essential for normal erythropoiesis and Epo receptor signaling. Blood. 122: 262-71
Maxwell MJ, Tsantikos E, Kong AM, et al. (2012) Attenuation of phosphoinositide 3-kinase δ signaling restrains autoimmune disease. Journal of Autoimmunity. 38: 381-91
Kim S, Kim SF, Maag D, et al. (2011) Amino acid signaling to mTOR mediated by inositol polyphosphate multikinase. Cell Metabolism. 13: 215-21
Maag D, Maxwell MJ, Hardesty DA, et al. (2011) Inositol polyphosphate multikinase is a physiologic PI3-kinase that activates Akt/PKB. Proceedings of the National Academy of Sciences of the United States of America. 108: 1391-6
Chakraborty A, Koldobskiy MA, Bello NT, et al. (2010) Inositol pyrophosphates inhibit Akt signaling, thereby regulating insulin sensitivity and weight gain. Cell. 143: 897-910
Verhagen AM, Wallace ME, Goradia A, et al. (2009) A kinase-dead allele of Lyn attenuates autoimmune disease normally associated with Lyn deficiency. Journal of Immunology (Baltimore, Md. : 1950). 182: 2020-9
Schoenwaelder SM, Ono A, Sturgeon S, et al. (2007) Identification of a unique co-operative phosphoinositide 3-kinase signaling mechanism regulating integrin alpha IIb beta 3 adhesive function in platelets. The Journal of Biological Chemistry. 282: 28648-58
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