Shuo-Chien Ling, Ph.D.
Affiliations: | 2005 | University of Illinois, Urbana-Champaign, Urbana-Champaign, IL |
Area:
Cellular Memory Mechanisms, Developmental Psychobiology, Fragile X syndromeGoogle:
"Shuo-Chien Ling"Mean distance: 14.74 (cluster 17) | S | N | B | C | P |
Cross-listing: Cell Biology Tree
Parents
Sign in to add mentor| Vladimir Gelfand | grad student | (Cell Biology Tree) | ||
| William T. Greenough | grad student | 2005 | UIUC | |
| (Messenger ribonucleoprotein (mRNP) complexes: Proteomic and functional analysis.) | ||||
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Publications
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| Wong P, Ho WY, Yen YC, et al. (2020) The vulnerability of motor and frontal cortex-dependent behaviors in mice expressing ALS-linked mutation in TDP-43. Neurobiology of Aging. 92: 43-60 |
| Ho WY, Chang JC, Tyan SH, et al. (2019) FUS-mediated dysregulation of Sema5a, an autism-related gene, in FUS mice with hippocampus-dependent cognitive deficits. Human Molecular Genetics |
| Ho WY, Ling SC. (2019) Elevated FUS levels by overriding its autoregulation produce gain-of-toxicity properties that disrupt protein and RNA homeostasis. Autophagy. 1-3 |
| Ho WY, Tai YK, Chang JC, et al. (2019) The ALS-FTD-linked gene product, C9orf72, regulates neuronal morphogenesis via autophagy. Autophagy |
| Wang J, Ho WY, Lim K, et al. (2018) Cell-autonomous requirement of TDP-43, an ALS/FTD signature protein, for oligodendrocyte survival and myelination. Proceedings of the National Academy of Sciences of the United States of America |
| López-Erauskin J, Tadokoro T, Baughn MW, et al. (2018) ALS/FTD-Linked Mutation in FUS Suppresses Intra-axonal Protein Synthesis and Drives Disease Without Nuclear Loss-of-Function of FUS. Neuron |
| Lu W, Lakonishok M, Serpinskaya AS, et al. (2018) Ooplasmic flow cooperates with transport and anchorage in oocyte posterior determination. The Journal of Cell Biology |
| Ling SC. (2018) Synaptic Paths to Neurodegeneration: The Emerging Role of TDP-43 and FUS in Synaptic Functions. Neural Plasticity. 2018: 8413496 |
| Bennett CL, Dastidar SG, Ling SC, et al. (2018) Senataxin mutations elicit motor neuron degeneration phenotypes and yield TDP-43 mislocalization in ALS4 mice and human patients. Acta Neuropathologica |
| Mitchell JC, Constable R, So E, et al. (2015) Wild type human TDP-43 potentiates ALS-linked mutant TDP-43 driven progressive motor and cortical neuron degeneration with pathological features of ALS. Acta Neuropathologica Communications. 3: 36 |