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Chia-Wei Lin

Affiliations: 
Massachusetts Institute of Technology, Cambridge, MA, United States 
Area:
Adult Neurogenesis and Synaptic Plasticity
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"Chia-Wei Lin"
Mean distance: 15.97 (cluster 11)
 		SNBCP
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Parents

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Tsung-Yu Chen research assistant MIT
Show-Ling Shyng research assistant MIT
Carlos Lois grad student MIT

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Publications

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Sim S, Antolin S, Lin CW, et al. (2013) Increased cell-intrinsic excitability induces synaptic changes in new neurons in the adult dentate gyrus that require Npas4. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 33: 7928-40
Lin CW, Sim S, Ainsworth A, et al. (2010) Genetically increased cell-intrinsic excitability enhances neuronal integration into adult brain circuits. Neuron. 65: 32-9
Kelsch W, Lin CW, Mosley CP, et al. (2009) A critical period for activity-dependent synaptic development during olfactory bulb adult neurogenesis. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 29: 11852-8
Kelsch W, Lin CW, Lois C. (2008) Sequential development of synapses in dendritic domains during adult neurogenesis. Proceedings of the National Academy of Sciences of the United States of America. 105: 16803-8
Kelsch W, Mosley CP, Lin CW, et al. (2007) Distinct mammalian precursors are committed to generate neurons with defined dendritic projection patterns. Plos Biology. 5: e300
Lin CW, Lin YW, Yan FF, et al. (2006) Kir6.2 mutations associated with neonatal diabetes reduce expression of ATP-sensitive K+ channels: implications in disease mechanism and sulfonylurea therapy. Diabetes. 55: 1738-46
Lin CW, Yan F, Shimamura S, et al. (2005) Membrane phosphoinositides control insulin secretion through their effects on ATP-sensitive K+ channel activity. Diabetes. 54: 2852-8
Yan FF, Lin CW, Cartier EA, et al. (2005) Role of ubiquitin-proteasome degradation pathway in biogenesis efficiency of {beta}-cell ATP-sensitive potassium channels. American Journal of Physiology. Cell Physiology. 289: C1351-9
Li Y, Yu WP, Lin CW, et al. (2005) Oxidation and reduction control of the inactivation gating of Torpedo ClC-0 chloride channels. Biophysical Journal. 88: 3936-45
Yan F, Lin CW, Weisiger E, et al. (2004) Sulfonylureas correct trafficking defects of ATP-sensitive potassium channels caused by mutations in the sulfonylurea receptor. The Journal of Biological Chemistry. 279: 11096-105
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