Philip Robert Lee
Affiliations: | National Institute of Health, Bethesda, MD, United States |
Area:
neuron-glial interactions, gene expressionGoogle:
"Philip Lee"Mean distance: 16.16 (cluster 6)
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Publications
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Lee PR, Fields RD. (2020) Activity-Dependent Gene Expression in Neurons. The Neuroscientist : a Review Journal Bringing Neurobiology, Neurology and Psychiatry. 1073858420943515 |
Iacobas DA, Iacobas S, Lee PR, et al. (2019) Coordinated Activity of Transcriptional Networks Responding to the Pattern of Action Potential Firing in Neurons. Genes. 10 |
Kato D, Wake H, Lee PR, et al. (2019) Motor learning requires myelination to reduce asynchrony and spontaneity in neural activity. Glia |
Dutta DJ, Woo DH, Lee PR, et al. (2018) Regulation of myelin structure and conduction velocity by perinodal astrocytes. Proceedings of the National Academy of Sciences of the United States of America |
Lee PR, Cohen JE, Iacobas DA, et al. (2017) Gene networks activated by specific patterns of action potentials in dorsal root ganglia neurons. Scientific Reports. 7: 43765 |
Bukalo O, Lee PR, Fields RD. (2016) BDNF mRNA Abundance Regulated by Antidromic Action Potentials and AP-LTD in Hippocampus. Neuroscience Letters |
Ravin R, Blank PS, Busse B, et al. (2016) Blast shockwaves propagate Ca(2+) activity via purinergic astrocyte networks in human central nervous system cells. Scientific Reports. 6: 25713 |
Wake H, Ortiz FC, Woo DH, et al. (2015) Nonsynaptic junctions on myelinating glia promote preferential myelination of electrically active axons. Nature Communications. 6: 7844 |
Wake H, Ortiz FC, Woo DH, et al. (2015) Nonsynaptic junctions on myelinating glia promote preferential myelination of electrically active axons Nature Communications. 6 |
Cohen JE, Lee PR, Fields RD. (2014) Systematic identification of 3'-UTR regulatory elements in activity-dependent mRNA stability in hippocampal neurons. Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences. 369 |