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M. J. Pinter

Affiliations: 
University of Göteborg, Gothenburg, Västra Götalands län, Sweden 
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"M. Pinter"
Mean distance: 15.35 (cluster 6)
 
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Stan T. Nakanishi grad student 1999-2005 Emory University School of Medicine
Jonathan F. Prather post-doc 2001-2003 Emory

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Publications

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Spaulding EL, Sleigh JN, Morelli KH, et al. (2016) Synaptic Deficits at Neuromuscular Junctions in Two Mouse Models of Charcot-Marie-Tooth Type 2d. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 36: 3254-67
Carrasco DI, Seburn KL, Pinter MJ. (2015) Altered terminal Schwann cell morphology precedes denervation in SOD1 mice. Experimental Neurology
Novak KR, Norman J, Mitchell JR, et al. (2015) Sodium channel slow inactivation as a therapeutic target for myotonia congenita. Annals of Neurology. 77: 320-32
Carrasco DI, Bichler EK, Rich MM, et al. (2012) Motor terminal degeneration unaffected by activity changes in SOD1(G93A) mice; a possible role for glycolysis. Neurobiology of Disease. 48: 132-40
Bullinger KL, Nardelli P, Pinter MJ, et al. (2011) Permanent central synaptic disconnection of proprioceptors after nerve injury and regeneration. II. Loss of functional connectivity with motoneurons. Journal of Neurophysiology. 106: 2471-85
Prather JF, Nardelli P, Nakanishi ST, et al. (2011) Recovery of proprioceptive feedback from nerve crush. The Journal of Physiology. 589: 4935-47
Carrasco DI, Bichler EK, Seburn KL, et al. (2010) Nerve terminal degeneration is independent of muscle fiber genotype in SOD1 mice. Plos One. 5: e9802
Bichler EK, Nakanishi ST, Wang QB, et al. (2007) Enhanced transmission at a spinal synapse triggered in vivo by an injury signal independent of altered synaptic activity. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 27: 12851-9
Bichler EK, Carrasco DI, Rich MM, et al. (2007) Rat motoneuron properties recover following reinnervation in the absence of muscle activity and evoked acetylcholine release. The Journal of Physiology. 585: 47-56
Wang X, Engisch KL, Teichert RW, et al. (2006) Prolongation of evoked and spontaneous synaptic currents at the neuromuscular junction after activity blockade is caused by the upregulation of fetal acetylcholine receptors. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 26: 8983-7
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