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Rochelle M. Hines

Affiliations: 
Neuroscience New England Medical (Tufts), Boston, MA, United States 
Area:
Inhibitory Synapses, GABA
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"Rochelle Hines"
Mean distance: 14.38 (cluster 6)
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Parents

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Jeffrey A. Kleim grad student New England Medical (Tufts)
Stephen J. Moss grad student New England Medical (Tufts)
Alaa El-Din El-Husseini grad student 2004-2009 UBC

Children

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Austin John Boren grad student University of Nevada, Las Vegas
Shannon Force grad student 2016- University of Nevada Las Vegas
Rachel Ali Rodriguez grad student 2016- University of Nevada Las Vegas

Collaborators

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Publications

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Hines DJ, Contreras A, Garcia B, et al. (2022) Human ARHGEF9 intellectual disability syndrome is phenocopied by a mutation that disrupts collybistin binding to the GABA receptor α2 subunit. Molecular Psychiatry. 27: 1275
Hines DJ, Contreras A, Garcia B, et al. (2022) Human ARHGEF9 intellectual disability syndrome is phenocopied by a mutation that disrupts collybistin binding to the GABA receptor α2 subunit. Molecular Psychiatry
Barker JS, Hines RM. (2020) Regulation of GABA Receptor Subunit Expression in Substance Use Disorders. International Journal of Molecular Sciences. 21
Nathanson AJ, Zhang Y, Smalley JL, et al. (2019) Identification of a Core Amino Acid Motif within the α Subunit of GABARs that Promotes Inhibitory Synaptogenesis and Resilience to Seizures. Cell Reports. 28: 670-681.e8
Contreras A, Hines DJ, Hines RM. (2019) Molecular Specialization of GABAergic Synapses on the Soma and Axon in Cortical and Hippocampal Circuit Function and Dysfunction. Frontiers in Molecular Neuroscience. 12: 154
Hines RM, Maric HM, Hines DJ, et al. (2018) Developmental seizures and mortality result from reducing GABA receptor α2-subunit interaction with collybistin. Nature Communications. 9: 3130
Ali Rodriguez R, Joya C, Hines RM. (2018) Common Ribs of Inhibitory Synaptic Dysfunction in the Umbrella of Neurodevelopmental Disorders. Frontiers in Molecular Neuroscience. 11: 132
Mircsof D, Langouët M, Rio M, et al. (2015) Mutations in NONO lead to syndromic intellectual disability and inhibitory synaptic defects. Nature Neuroscience
Silayeva L, Deeb TZ, Hines RM, et al. (2015) KCC2 activity is critical in limiting the onset and severity of status epilepticus. Proceedings of the National Academy of Sciences of the United States of America. 112: 3523-8
Engin E, Bakhurin KI, Smith KS, et al. (2014) Neural basis of benzodiazepine reward: requirement for α2 containing GABAA receptors in the nucleus accumbens. Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology. 39: 1805-15
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