Jan M. Schulz, PhD
Affiliations: | Physiology | University of Bern, Bern, Bern, Switzerland |
Area:
Striatum, cortex, synaptic integrationGoogle:
"Jan Schulz"Mean distance: 14.72 (cluster 6) | S | N | B | C | P |
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Publications
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| Traunmüller L, Schulz J, Ortiz R, et al. (2023) A cell-type-specific alternative splicing regulator shapes synapse properties in a trans-synaptic manner. Cell Reports. 42: 112173 |
| Kay JW, Schulz JM, Phillips WA. (2022) A Comparison of Partial Information Decompositions Using Data from Real and Simulated Layer 5b Pyramidal Cells. Entropy (Basel, Switzerland). 24 |
| Lodge M, Hernandez MC, Schulz JM, et al. (2021) Sparsification of AP firing in adult-born hippocampal granule cells via voltage-dependent α5-GABA receptors. Cell Reports. 37: 109768 |
| Schulz JM, Kay JW, Bischofberger J, et al. (2021) GABA Receptor-Mediated Regulation of Dendro-Somatic Synergy in Layer 5 Pyramidal Neurons. Frontiers in Cellular Neuroscience. 15: 718413 |
| Boddington LJ, Gray JP, Schulz JM, et al. (2019) Low-intensity contralesional electrical theta burst stimulation modulates ipsilesional excitability and enhances stroke recovery. Experimental Neurology. 113071 |
| Schulz JM, Knoflach F, Hernandez MC, et al. (2019) Enhanced dendritic inhibition and impaired NMDAR activation in a mouse model of Down syndrome. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience |
| Schulz JM, Knoflach F, Hernandez MC, et al. (2018) Dendrite-targeting interneurons control synaptic NMDA-receptor activation via nonlinear α5-GABA receptors. Nature Communications. 9: 3576 |
| Schulz JM, Redgrave P, Reynolds JN. (2015) Corrigendum: Cortico-striatal spike-timing dependent plasticity after activation of subcortical pathways. Frontiers in Synaptic Neuroscience. 7: 13 |
| Oswald MJ, Schulz JM, Kelsch W, et al. (2015) Potentiation of NMDA receptor-mediated transmission in striatal cholinergic interneurons. Frontiers in Cellular Neuroscience. 9: 116 |
| Oettinghaus B, Schulz JM, Restelli LM, et al. (2015) Synaptic dysfunction, memory deficits and hippocampal atrophy due to ablation of mitochondrial fission in adult forebrain neurons. Cell Death and Differentiation |