Soham Chanda
Affiliations: | 2019- | Biochemistry & Molecular Biology | Colorado State University, Fort Collins, CO |
Area:
Synaptic PlasticityGoogle:
"Soham Chanda"Mean distance: 14.02 (cluster 6) | S | N | B | C | P |
Parents
Sign in to add mentorMatthew A. Xu-Friedman | grad student | 2007-2010 | SUNY Buffalo |
Thomas C. Sudhof | post-doc | 2011-2018 | Stanford |
BETA: Related publications
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Publications
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Tahtamouni LH, Alderfer SA, Kuhn TB, et al. (2023) Characterization of a Human Neuronal Culture System for the Study of Cofilin-Actin Rod Pathology. Biomedicines. 11 |
Benner O, Cast TP, Minamide LS, et al. (2023) Multiple N-linked glycosylation sites critically modulate the synaptic abundance of neuroligin isoforms. The Journal of Biological Chemistry. 299: 105361 |
Haddad Derafshi B, Danko T, Chanda S, et al. (2022) The autism risk factor CHD8 is a chromatin activator in human neurons and functionally dependent on the ERK-MAPK pathway effector ELK1. Scientific Reports. 12: 22425 |
Burlingham SR, Wong NF, Peterkin L, et al. (2022) Induction of synapse formation by de novo neurotransmitter synthesis. Nature Communications. 13: 3060 |
Ng YH, Chanda S, Janas JA, et al. (2021) Efficient generation of dopaminergic induced neuronal cells with midbrain characteristics. Stem Cell Reports |
Ng YH, Chanda S, Janas JA, et al. (2021) Efficient generation of dopaminergic induced neuronal cells with midbrain characteristics. Stem Cell Reports |
Cast T, Boesch D, Smyth K, et al. (2020) An Autism-Associated Mutation Impairs Neuroligin-4 Glycosylation and Enhances Excitatory Synaptic Transmission in Human Neurons. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience |
Lee QY, Mall M, Chanda S, et al. (2020) Pro-neuronal activity of Myod1 due to promiscuous binding to neuronal genes. Nature Cell Biology |
Marro SG, Chanda S, Yang N, et al. (2019) Neuroligin-4 Regulates Excitatory Synaptic Transmission in Human Neurons. Neuron |
Chanda S, Ang CE, Lee QY, et al. (2019) Direct Reprogramming of Human Neurons Identifies MARCKSL1 as a Pathogenic Mediator of Valproic Acid-Induced Teratogenicity. Cell Stem Cell |