Jack de la Torre, MD, PhD

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Obrenovich ME, Smith MA, Siedlak SL, et al. (2006) Overexpression of GRK2 in Alzheimer disease and in a chronic hypoperfusion rat model is an early marker of brain mitochondrial lesions. Neurotoxicity Research. 10: 43-56
Aliyev A, Chen SG, Seyidova D, et al. (2005) Mitochondria DNA deletions in atherosclerotic hypoperfused brain microvessels as a primary target for the development of Alzheimer's disease. Journal of the Neurological Sciences. 229: 285-92
Liu Q, Smith MA, AvilÃ¡ J, et al. (2005) Alzheimer-specific epitopes of tau represent lipid peroxidation-induced conformations. Free Radical Biology & Medicine. 38: 746-54
Aliev G, Smith MA, de la Torre JC, et al. (2004) Mitochondria as a primary target for vascular hypoperfusion and oxidative stress in Alzheimer's disease. Mitochondrion. 4: 649-63
de la Torre J, Aliev G, Perry G. (2004) Drug therapy in Alzheimer's disease. The New England Journal of Medicine. 351: 1911-3; author reply
Seyidova D, Aliyev A, Rzayev N, et al. (2004) The role of nitric oxide in the pathogenesis of brain lesions during the development of Alzheimer's disease. In Vivo (Athens, Greece). 18: 325-33
Aliyev A, Seyidova D, Rzayev N, et al. (2004) Is nitric oxide a key target in the pathogenesis of brain lesions during the development of Alzheimer's disease? Neurological Research. 26: 547-53
Aliev G, Smith MA, Obrenovich ME, et al. (2003) Role of vascular hypoperfusion-induced oxidative stress and mitochondria failure in the pathogenesis of Azheimer disease. Neurotoxicity Research. 5: 491-504