Christopher M. Evans
Affiliations: | Pulmonary and Critical Care Medicine | University of Colorado School of Medicine, Aurora, CO, United States |
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Parents
Sign in to add mentor| Allison D. Fryer | grad student | 1996-2000 | Johns Hopkins | |
| (Airway hyperreactivity in guinea pigs: Interactions between eosinophils and airway nerves.) | ||||
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| Singanayagam A, Footitt J, Marczynski M, et al. (2022) Airway mucins promote immunopathology in virus-exacerbated chronic obstructive pulmonary disease. The Journal of Clinical Investigation |
| Fakih DC, Rodríguez-Piñeiro AM, Trillo-Muyo S, et al. (2020) Normal murine respiratory tract has its mucus concentrated in clouds based on the Muc5b mucin. American Journal of Physiology. Lung Cellular and Molecular Physiology |
| Jaramillo AM, Evans CM. (2019) It Takes 1 for Type 2: IL-1 Receptor Mediates Eosinophilia in Scnn1b Transgenic Mice. American Journal of Respiratory Cell and Molecular Biology |
| Campbell L, Hepworth MR, Whittingham-Dowd J, et al. (2019) ILC2s mediate systemic innate protection by priming mucus production at distal mucosal sites. The Journal of Experimental Medicine |
| Parimon T, Yao C, Habiel DM, et al. (2019) Syndecan-1 promotes lung fibrosis by regulating epithelial reprogramming through extracellular vesicles. Jci Insight. 5 |
| Svedberg FR, Brown SL, Krauss MZ, et al. (2019) The lung environment controls alveolar macrophage metabolism and responsiveness in type 2 inflammation. Nature Immunology |
| Jaramillo AM, Piccotti L, Velasco WV, et al. (2019) Different Munc18 proteins mediate baseline and stimulated airway mucin secretion. Jci Insight |
| Hancock LA, Hennessy CE, Solomon GM, et al. (2018) Muc5b overexpression causes mucociliary dysfunction and enhances lung fibrosis in mice. Nature Communications. 9: 5363 |
| Benam KH, Vladar EK, Janssen WJ, et al. (2018) Mucociliary Defense: Emerging Cellular, Molecular, and Animal Models. Annals of the American Thoracic Society. 15: S210-S215 |
| De la Garza MM, Cumpian AM, Daliri S, et al. (2018) COPD-Type lung inflammation promotes K-ras mutant lung cancer through epithelial HIF-1α mediated tumor angiogenesis and proliferation. Oncotarget. 9: 32972-32983 |