Yingbin Fu, Ph.D.

Affiliations: 
University of Utah, Salt Lake City, UT 
Area:
Visual system
Website:
http://uuhsc.utah.edu/MoranEyeCenter/research/faculty/primary/fu_lab.htm
Google:
"Yingbin Fu"
Mean distance: 14.46 (cluster 31)
 
SNBCP
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Publications

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Xu H, Jin N, Chuang JZ, et al. (2022) Visual pigment-deficient cones survive and mediate visual signaling despite the lack of outer segments. Proceedings of the National Academy of Sciences of the United States of America. 119
Xu H, Enemchukwu N, Zhong X, et al. (2020) Deletion of M-opsin prevents "M cone" degeneration in a mouse model of Leber congenital amaurosis. The American Journal of Pathology
Kumar S, Nakashizuka H, Jones A, et al. (2017) Proteolytic Degradation and Inflammation Play Critical Roles in Polypoidal Choroidal Vasculopathy. The American Journal of Pathology
Zhang T, Cao LH, Kumar S, et al. (2016) Dimerization of visual pigments in vivo. Proceedings of the National Academy of Sciences of the United States of America
Zhang T, Enemchukwu NO, Jones A, et al. (2015) Genetic deletion of S-opsin prevents rapid cone degeneration in a mouse model of Leber congenital amaurosis. Human Molecular Genetics. 24: 1755-63
Fu Y. (2014) A functional approach to examine the role of HTRA1 versus ARMS2 in AMD. Investigative Ophthalmology & Visual Science. 55: 6524
Kumar S, Berriochoa Z, Ambati BK, et al. (2014) Angiographic features of transgenic mice with increased expression of human serine protease HTRA1 in retinal pigment epithelium. Investigative Ophthalmology & Visual Science. 55: 3842-50
Fu Y, Zhang T. (2014) Pathophysilogical mechanism and treatment strategies for Leber congenital amaurosis. Advances in Experimental Medicine and Biology. 801: 791-6
Kumar S, Berriochoa Z, Jones AD, et al. (2014) Detecting abnormalities in choroidal vasculature in a mouse model of age-related macular degeneration by time-course indocyanine green angiography. Journal of Visualized Experiments : Jove. e51061
Luo L, Uehara H, Zhang X, et al. (2013) Photoreceptor avascular privilege is shielded by soluble VEGF receptor-1. Elife. 2: e00324
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