Jeehyun Kwag, D.Phil

Affiliations: 
Brain and Cognitive Sciences Seoul National University, Seoul, South Korea 
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"Jeehyun Kwag"
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Publications

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Park K, Kohl MM, Kwag J. (2023) Memory encoding and retrieval by retrosplenial parvalbumin interneurons are impaired in Alzheimer's disease model mice. Current Biology : Cb
Toi PT, Jang HJ, Min K, et al. (2022) In vivo direct imaging of neuronal activity at high temporospatial resolution. Science (New York, N.Y.). 378: 160-168
Lee J, Kwag J. (2021) Activation of PLCβ1 enhances endocannabinoid mobilization to restore hippocampal spike-timing-dependent potentiation and contextual fear memory impaired by Alzheimer's amyloidosis. Alzheimer's Research & Therapy. 13: 165
Prince LY, Tran MM, Grey D, et al. (2021) Neocortical inhibitory interneuron subtypes are differentially attuned to synchrony- and rate-coded information. Communications Biology. 4: 935
Gwak J, Kwag J. (2020) Distinct subtypes of inhibitory interneurons differentially promote the propagation of rate and temporal codes in the feedforward neural network. Chaos (Woodbury, N.Y.). 30: 053102
Jang HJ, Chung H, Rowland JM, et al. (2020) Distinct roles of parvalbumin and somatostatin interneurons in gating the synchronization of spike times in the neocortex. Science Advances. 6: eaay5333
Chung H, Park K, Jang HJ, et al. (2020) Dissociation of somatostatin and parvalbumin interneurons circuit dysfunctions underlying hippocampal theta and gamma oscillations impaired by amyloid β oligomers in vivo. Brain Structure & Function
Park K, Lee J, Jang HJ, et al. (2020) Optogenetic activation of parvalbumin and somatostatin interneurons selectively restores theta-nested gamma oscillations and oscillation-induced spike timing-dependent long-term potentiation impaired by amyloid β oligomers. Bmc Biology. 18: 7
Park SW, Jang HJ, Kim M, et al. (2019) Spatiotemporally random and diverse grid cell spike patterns contribute to the transformation of grid cell to place cell in a neural network model. Plos One. 14: e0225100
Lee J, Kwag J. (2019) Aβ oligomers impair mGluR5-mediated enhancement of depolarization-induced suppression of inhibition by disrupting PLCβ-dependent endocannabinoid mobilization Ibro Reports. 6
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