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Dai Horiuchi, Ph.D.

Affiliations: 
2007 Molecular, Cellular, and Developmental Biology Indiana University, Bloomington, Bloomington, IN, United States 
Area:
Cell Biology, Genetics, Neuroscience Biology
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"Dai Horiuchi"
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William M. Saxton grad student 2007 Indiana University
 (Studies on the functions of APLIP1, a Drosophila JNK pathway scaffold protein, in axonal transport.)

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Publications

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Kang HJ, Song HY, Guo Y, et al. (2018) NQO1 regulates mitotic progression and response to mitotic stress through modulating SIRT2 activity. Free Radical Biology & Medicine
Horiuchi D, Zhou AY, Corella AN, et al. (2016) Abstract B34: PIM kinase as a novel therapeutic target for triple-negative breast cancer Molecular Cancer Research. 14
Martins MM, Zhou AY, Corella A, et al. (2015) Linking tumor mutations to drug responses via a quantitative chemical-genetic interaction map. Cancer Discovery. 5: 154-67
Martins MM, Zhou AY, Corella A, et al. (2015) Abstract PR15: Functional analysis of diverse oncogenic driver mutations using an isogenic cell line library identifies novel drug responses and alterations in metabolism Cancer Research. 75
Martins MM, Zhou AY, Corella A, et al. (2015) Abstract PR07: Functional analysis of diverse oncogenic driver mutations using an isogenic cell line library identifies novel drug responses and alterations in metabolism Cancer Research. 75
Horiuchi D, Zhou AY, Corella AN, et al. (2014) Abstract LB-122: PIM1 kinase inhibition halts the growth of MYC-overexpressing triple-negative breast tumors Cancer Research. 74
Goga A, Martins M, Corella A, et al. (2013) Abstract P5-10-01: A systems approach to combine genomics and emerging therapeutics to discover new treatments for TNBC Cancer Research. 73
Horiuchi D, Huskey NE, Kusdra L, et al. (2012) Chemical-genetic analysis of cyclin dependent kinase 2 function reveals an important role in cellular transformation by multiple oncogenic pathways. Proceedings of the National Academy of Sciences of the United States of America. 109: E1019-27
Merrick KA, Wohlbold L, Zhang C, et al. (2011) Switching Cdk2 on or off with small molecules to reveal requirements in human cell proliferation. Molecular Cell. 42: 624-36
Huskey NE, Horiuchi D, Kusdra L, et al. (2009) Abstract C68: Using a chemical genetic approach to define the role of CDK2 in normal and transformed cell lines Cancer Research. 69
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