Mohamed Chahine

Affiliations: 
Universite Laval (Canada) 
Area:
Medical Biophysics, General Biophysics, Neuroscience Biology
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"Mohamed Chahine"
Mean distance: 53433
 
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Publications

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Chapotte-Baldacci CA, Pierre M, Djemai M, et al. (2023) Biophysical properties of Na1.5 channels from atrial-like and ventricular-like cardiomyocytes derived from human induced pluripotent stem cells. Scientific Reports. 13: 20685
Pierre M, Djemai M, Chapotte-Baldacci CA, et al. (2023) Cardiac involvement in patient-specific induced pluripotent stem cells of myotonic dystrophy type 1: unveiling the impact of voltage-gated sodium channels. Frontiers in Physiology. 14: 1258318
Plumereau Q, Ebdalla A, Poulin H, et al. (2022) De novo Y1460C missense variant in Na1.1 impedes the pore region and results in epileptic encephalopathy. Scientific Reports. 12: 17182
Gamal El-Din TM, Zimmer T, Chahine M. (2022) Editorial: Structure Related Druggability of Voltage-Gated Sodium and Calcium Ion-Channels to Treat Diseases. Frontiers in Pharmacology. 13: 947511
Yang XR, Ginjupalli VKM, Theriault O, et al. (2022) -related epilepsy of infancy with migrating focal seizures: report of a variant with apparent gain- and loss-of-function effects. Journal of Neurophysiology. 127: 1388-1397
Hui JB, Silva JCH, Pelaez MC, et al. (2022) NPRL2 Inhibition of mTORC1 Controls Sodium Channel Expression and Brain Amino Acid Homeostasis. Eneuro. 9
Pierre M, Djemai M, Poulin H, et al. (2021) Na1.5 knockout in iPSCs: a novel approach to study Na1.5 variants in a human cardiomyocyte environment. Scientific Reports. 11: 17168
Plumereau Q, Theriault O, Pouliot V, et al. (2021) Novel G1481V and Q1491H SCN5A Mutations Linked to Long QT Syndrome Destabilize the Nav1.5 Inactivation State. Cjc Open. 3: 256-266
Poulin H, Mercier A, Djemai M, et al. (2021) iPSC-derived cardiomyocytes from patients with myotonic dystrophy type 1 have abnormal ion channel functions and slower conduction velocities. Scientific Reports. 11: 2500
Poulin H, Chahine M. (2021) R1617Q epilepsy mutation slows Na 1.6 sodium channel inactivation and increases the persistent current and neuronal firing. The Journal of Physiology
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