Gopal Thinakaran
Affiliations: | University of Chicago, Chicago, IL |
Area:
Cell Biology, Neuroscience BiologyGoogle:
"Gopal Thinakaran"Mean distance: 106866
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Publications
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| Aow J, Huang TR, Goh YT, et al. (2023) Evidence for a clathrin-independent endocytic pathway for APP internalization in the neuronal somatodendritic compartment. Cell Reports. 42: 112774 |
| Ulku I, Liebsch F, Akerman SC, et al. (2023) Mechanisms of amyloid-β34 generation indicate a pivotal role for BACE1 in amyloid homeostasis. Scientific Reports. 13: 2216 |
| Ponnusamy M, Wang S, Yuksel M, et al. (2022) Loss of forebrain BIN1 attenuates hippocampal pathology and neuroinflammation in a tauopathy model. Brain : a Journal of Neurology |
| Sudwarts A, Ramesha S, Gao T, et al. (2022) BIN1 is a key regulator of proinflammatory and neurodegeneration-related activation in microglia. Molecular Neurodegeneration. 17: 33 |
| Aow J, Huang TR, Thinakaran G, et al. (2022) Enhanced cleavage of APP by co-expressed Bace1 alters the distribution of APP and its fragments in neuronal and non-neuronal cells. Molecular Neurobiology |
| De Rossi P, Nomura T, Andrew RJ, et al. (2020) Neuronal BIN1 Regulates Presynaptic Neurotransmitter Release and Memory Consolidation. Cell Reports. 30: 3520-3535.e7 |
| Deyts C, Clutter M, Pierce N, et al. (2019) APP-Mediated Signaling Prevents Memory Decline in Alzheimer's Disease Mouse Model. Cell Reports. 27: 1345-1355.e6 |
| Zerweck J, Venkata BS, Pittman JM, et al. (2019) A versatile method for producing labeled or unlabeled Aβ55, Aβ40, and other β-amyloid family peptides. Protein Expression and Purification |
| Andrew RJ, De Rossi P, Nguyen P, et al. (2019) Reduction of the expression of the late-onset Alzheimer's disease (AD) risk-factor BIN1 does not affect amyloid pathology in an AD mouse model. The Journal of Biological Chemistry |
| De Rossi P, Andrew RJ, Musial TF, et al. (2018) Aberrant accrual of BIN1 near Alzheimer's disease amyloid deposits in transgenic models. Brain Pathology (Zurich, Switzerland) |