Ji-Yeon Shin, Ph.D.
Affiliations: | 2006 | Emory University, Atlanta, GA |
Area:
Neuroscience Biology, Molecular Biology, Genetics, PathologyGoogle:
"Ji-Yeon Shin"Mean distance: 19430.2
Parents
Sign in to add mentor| Xiao-Jiang Li | grad student | 2006 | Emory | |
| (Expression of mutant huntingtin in glial cells contributes to the neuropathology of Huntington's disease.) | ||||
| Howard J Worman | post-doc | Columbia University Medical School (Cell Biology Tree) | ||
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Publications
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| Hernandez-Ono A, Zhao YP, Murray JW, et al. (2024) Dynamic regulation of hepatic lipid metabolism by torsinA and its activators. Jci Insight |
| Hernandez-Ono A, Zhao YP, Murray JW, et al. (2023) Functional interaction of torsinA and its activators in liver lipid metabolism. Biorxiv : the Preprint Server For Biology |
| Östlund C, Hernandez-Ono A, Turk SJ, et al. (2022) Hepatocytes deficient in nuclear envelope protein lamina-associated polypeptide 1 are an ideal mammalian system to study intranuclear lipid droplets. Journal of Lipid Research. 100277 |
| Wang Y, Shilagardi K, Hsu T, et al. (2022) Abolishing the prelamin A ZMPSTE24 cleavage site leads to progeroid phenotypes with near-normal longevity in mice. Proceedings of the National Academy of Sciences of the United States of America. 119 |
| Shin JY, Worman HJ. (2021) Molecular Pathology of Laminopathies. Annual Review of Pathology |
| Kronenberg-Tenga R, Tatli M, Eibauer M, et al. (2021) A lamin A/C variant causing striated muscle disease provides insights into filament organization. Journal of Cell Science |
| Shin JY, Hernandez-Ono A, Fedotova T, et al. (2019) Nuclear envelope-localized torsinA-LAP1 complex regulates hepatic VLDL secretion and steatosis. The Journal of Clinical Investigation. 130 |
| Wang Y, Shin JY, Nakanishi K, et al. (2019) Postnatal Development of Mice with Combined Genetic Depletions of Lamin A/C, Emerin and Lamina-associated Polypeptide 1. Human Molecular Genetics |
| Wu W, Chordia MD, Hart BP, et al. (2016) Macrocyclic MEK1/2 inhibitor with efficacy in a mouse model of cardiomyopathy caused by lamin A/C gene mutation. Bioorganic & Medicinal Chemistry |
| Shin JY, Méndez-López I, Hong M, et al. (2016) Lamina-associated polypeptide 1 is dispensable for embryonic myogenesis but required for postnatal skeletal muscle growth. Human Molecular Genetics |