Paul D. Coleman

Affiliations: 
University of Rochester Medical Center, Rochester, NY, United States 
Area:
Alzheimer's disease
Google:
"Paul Coleman"
Mean distance: 14.72 (cluster 19)
 
Cross-listing: Alzheimer's Tree

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Publications

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Brokaw DL, Piras IS, Mastroeni D, et al. (2020) Cell death and survival pathways in Alzheimer's disease: an integrative hypothesis testing approach utilizing -omic data sets. Neurobiology of Aging. 95: 15-25
Deng W, Xing C, David R, et al. (2019) AmpliSeq Transcriptome of Laser Captured Neurons from Alzheimer Brain: Comparison of Single Cell Versus Neuron Pools. Aging and Disease. 10: 1146-1158
Lardenoije R, Roubroeks JAY, Pishva E, et al. (2019) Alzheimer's disease-associated (hydroxy)methylomic changes in the brain and blood. Clinical Epigenetics. 11: 164
Piras IS, Krate J, Delvaux E, et al. (2019) Transcriptome Changes in the Alzheimer's Disease Middle Temporal Gyrus: Importance of RNA Metabolism and Mitochondria-Associated Membrane Genes. Journal of Alzheimer's Disease : Jad
Smith RG, Pishva E, Shireby G, et al. (2019) O4‐10‐06: Integrated Genetic‐Epigenetic Analyses Of Alzheimer'S Disease Alzheimers & Dementia. 15
Mastroeni D, Nolz J, Sekar S, et al. (2017) Laser-captured microglia in the Alzheimer's and Parkinson's brain reveal unique regional expression profiles and suggest a potential role for hepatitis B in the Alzheimer's brain. Neurobiology of Aging. 63: 12-21
Delvaux E, Mastroeni D, Nolz J, et al. (2017) Multivariate analyses of peripheral blood leukocyte transcripts distinguish Alzheimer's, Parkinson's, control, and those at risk for developing Alzheimer's. Neurobiology of Aging
Mastroeni D, Sekar S, Nolz J, et al. (2017) ANK1 is up-regulated in laser captured microglia in Alzheimer's brain; the importance of addressing cellular heterogeneity. Plos One. 12: e0177814
Coleman PD, Mastroeni D. (2017) A call for new approaches to Alzheimer's disease research. Neurobiology of Aging
Mastroeni D, Khdour OM, Delvaux E, et al. (2016) Nuclear but not mitochondrial-encoded OXPHOS genes are altered in aging, mild cognitive impairment, and Alzheimer's disease. Alzheimer's & Dementia : the Journal of the Alzheimer's Association
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