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Susan Knoblach

Affiliations: 
Biomedical Sciences The George Washington University, Washington, DC, United States 
Area:
Neuroscience Biology
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"Susan Knoblach"
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Bruce Lerman grad student 2010 The George Washington University

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Darnall RA, Chen X, Nemani KV, et al. (2017) Early post-natal exposure to intermittent hypoxia in rodents is pro-inflammatory, impairs white matter integrity and alters brain metabolism. Pediatric Research
Nalbandian A, Ghimbovschi S, Wang Z, et al. (2015) Global gene expression profiling in R155H knock-in murine model of VCP disease. Clinical and Translational Science. 8: 8-16
Yazdi PG, Su H, Ghimbovschi S, et al. (2013) Differential gene expression reveals mitochondrial dysfunction in an imprinting center deletion mouse model of Prader-Willi syndrome. Clinical and Translational Science. 6: 347-55
Lerman BJ, Hoffman EP, Sutherland ML, et al. (2012) Deletion of galectin-3 exacerbates microglial activation and accelerates disease progression and demise in a SOD1(G93A) mouse model of amyotrophic lateral sclerosis. Brain and Behavior. 2: 563-75
Pajoohesh-Ganji A, Knoblach SM, Faden AI, et al. (2012) Characterization of inflammatory gene expression and galectin-3 function after spinal cord injury in mice. Brain Research. 1475: 96-105
Kesari A, Fukuda M, Knoblach S, et al. (2008) Dysferlin deficiency shows compensatory induction of Rab27A/Slp2a that may contribute to inflammatory onset. The American Journal of Pathology. 173: 1476-87
Spurney CF, Knoblach S, Pistilli EE, et al. (2008) Dystrophin-deficient cardiomyopathy in mouse: expression of Nox4 and Lox are associated with fibrosis and altered functional parameters in the heart. Neuromuscular Disorders : Nmd. 18: 371-81
Byrnes K, Stoica B, Giovanni SD, et al. (2006) The Role of Microglia and NADPH Oxidase in Traumatic Spinal Cord Injury Neurorx. 3: 408-408
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