Nathan D. Pankratz, Ph.D.
Affiliations: | 2003 | Indiana University, Bloomington, Bloomington, IN, United States |
Area:
Genetics, PathologyGoogle:
"Nathan Pankratz"Mean distance: 42746.4
Parents
Sign in to add mentor| Tatiana Foroud | grad student | 2003 | Indiana University Bloomington | |
| (Study to identify susceptibility genes for Parkinson disease.) | ||||
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Publications
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| Pankratz N, Cole BR, Beutel KM, et al. (2023) Parkinson Disease Genetics Extended to African and Hispanic Ancestries in the VA Million Veteran Program. Neurology. Genetics. 10: e200110 |
| Huffman JE, Nicolas J, Hahn J, et al. (2023) Whole genome analysis of plasma fibrinogen reveals population-differentiated genetic regulators with putative liver roles. Medrxiv : the Preprint Server For Health Sciences |
| Pankratz N, Wei P, Brody JA, et al. (2022) Whole exome sequencing of 14 389 individuals from the ESP and CHARGE consortia identifies novel rare variation associated with hemostatic factors. Human Molecular Genetics |
| Taub MA, Conomos MP, Keener R, et al. (2022) Genetic determinants of telomere length from 109,122 ancestrally diverse whole-genome sequences in TOPMed. Cell Genomics. 2 |
| Mikhaylova AV, McHugh CP, Polfus LM, et al. (2021) Whole-genome sequencing in diverse subjects identifies genetic correlates of leukocyte traits: The NHLBI TOPMed program. American Journal of Human Genetics |
| Hu Y, Stilp AM, McHugh CP, et al. (2021) Whole-genome sequencing association analysis of quantitative red blood cell phenotypes: The NHLBI TOPMed program. American Journal of Human Genetics |
| Vuckovic D, Bao EL, Akbari P, et al. (2020) The Polygenic and Monogenic Basis of Blood Traits and Diseases. Cell. 182: 1214-1231.e11 |
| Chen MH, Raffield LM, Mousas A, et al. (2020) Trans-ethnic and Ancestry-Specific Blood-Cell Genetics in 746,667 Individuals from 5 Global Populations. Cell. 182: 1198-1213.e14 |
| Martínez de LaPiscina I, Hernández-Ramírez LC, Portillo N, et al. (2020) Rare Germline Variants in Pediatric Patients With Cushing's Disease: What Is Their Role? Frontiers in Endocrinology. 11: 433 |
| Chasseloup F, Pankratz N, Lane J, et al. (2020) Germline CDKN1B loss-of-function variants cause pediatric Cushing's disease with or without an MEN4 phenotype. The Journal of Clinical Endocrinology and Metabolism |