Michael Tal
Affiliations: | Medical Neurobiology | Hebrew University, Schools of Dentistry and Medicine Jerusalem |
Area:
Mechanisms of chronic painGoogle:
"Michael Tal"Mean distance: 15.28 (cluster 31) | S | N | B | C | P |
Parents
Sign in to add mentor
BETA: Related publications
See more...
Publications
|
You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect. |
| Devor M, Tal M. (2014) Nerve resection for the treatment of chronic neuropathic pain. Pain. 155: 1053-4 |
| Nitzan-Luques A, Minert A, Devor M, et al. (2013) Dynamic genotype-selective "phenotypic switching" of CGRP expression contributes to differential neuropathic pain phenotype. Experimental Neurology. 250: 194-204 |
| Ringkamp M, Tal M, Hartke TV, et al. (2012) Local loperamide injection reduces mechanosensitivity of rat cutaneous, nociceptive C-fibers. Plos One. 7: e42105 |
| Nitzan-Luques A, Devor M, Tal M. (2011) Genotype-selective phenotypic switch in primary afferent neurons contributes to neuropathic pain. Pain. 152: 2413-26 |
| Gabay E, Wolf G, Shavit Y, et al. (2011) Chronic blockade of interleukin-1 (IL-1) prevents and attenuates neuropathic pain behavior and spontaneous ectopic neuronal activity following nerve injury. European Journal of Pain (London, England). 15: 242-8 |
| Nissenbaum J, Devor M, Seltzer Z, et al. (2010) Susceptibility to chronic pain following nerve injury is genetically affected by CACNG2. Genome Research. 20: 1180-90 |
| Eliav E, Benoliel R, Herzberg U, et al. (2009) The role of IL-6 and IL-1beta in painful perineural inflammatory neuritis. Brain, Behavior, and Immunity. 23: 474-84 |
| Devor M, Tal M. (2009) What causes low back pain? Pain. 142: 11-2 |
| Kaufmann D, Yagen B, Minert A, et al. (2008) Evaluation of the enantioselective antiallodynic and pharmacokinetic profile of propylisopropylacetamide, a chiral isomer of valproic acid amide. Neuropharmacology. 54: 699-707 |
| Kleibeuker W, Gabay E, Kavelaars A, et al. (2008) IL-1 beta signaling is required for mechanical allodynia induced by nerve injury and for the ensuing reduction in spinal cord neuronal GRK2. Brain, Behavior, and Immunity. 22: 200-8 |