Gennadij Raivich, M.D. D.Sc.
Affiliations: | Perinatal Brain Repair | University College London, London, United Kingdom |
Area:
Hypoxic Ischemic EncephalopathyWebsite:
http://iris.ucl.ac.uk/iris/Google:
"Gennadij Raivich"Mean distance: 15.44 (cluster 3) | S | N | B | C | P |
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Publications
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| Thei L, Rocha-Ferreira E, Peebles D, et al. (2018) Extracellular Signal-Related Kinase (ERK) 2 has duality in function between neuronal and astrocyte expression following neonatal hypoxia-ischemic cerebral injury. The Journal of Physiology |
| Hristova M, Rocha-Ferreira E, Fontana X, et al. (2015) Inhibition of Signal Transducer and Activator of Transcription 3 (STAT3) reduces neonatal hypoxic-ischemic brain damage. Journal of Neurochemistry |
| Lange S, Rocha-Ferreira E, Thei L, et al. (2014) Peptidylarginine deiminases: novel drug targets for prevention of neuronal damage following hypoxic ischemic insult (HI) in neonates. Journal of Neurochemistry. 130: 555-62 |
| Arthur-Farraj PJ, Latouche M, Wilton DK, et al. (2012) c-Jun reprograms Schwann cells of injured nerves to generate a repair cell essential for regeneration. Neuron. 75: 633-47 |
| Fontana X, Hristova M, Da Costa C, et al. (2012) c-Jun in Schwann cells promotes axonal regeneration and motoneuron survival via paracrine signaling. The Journal of Cell Biology. 198: 127-41 |
| Patodia S, Raivich G. (2012) Downstream effector molecules in successful peripheral nerve regeneration. Cell and Tissue Research. 349: 15-26 |
| Ruff CA, Staak N, Patodia S, et al. (2012) Neuronal c-Jun is required for successful axonal regeneration, but the effects of phosphorylation of its N-terminus are moderate. Journal of Neurochemistry. 121: 607-18 |
| Patodia S, Raivich G. (2012) Role of transcription factors in peripheral nerve regeneration. Frontiers in Molecular Neuroscience. 5: 8 |
| Hunt D, Raivich G, Anderson PN. (2012) Activating transcription factor 3 and the nervous system. Frontiers in Molecular Neuroscience. 5: 7 |
| Sahota A, Kendall G, Lange S, et al. (2012) TNF gene deletion prevents lipopolysaccharide-mediated sensitisation of the neonatal mouse brain to hypoxic-ischaemic insult Bmc Proceedings. 6: 1-1 |