Avi Ashkenazi
Affiliations: | Genentech, Inc., San Francisco, CA, United States |
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Publications
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| Harnoss JM, Le Thomas A, Reichelt M, et al. (2020) IRE1α disruption in triple-negative breast cancer cooperates with anti-angiogenic therapy by reversing ER stress adaptation and remodeling the tumor microenvironment. Cancer Research |
| Lam M, Marsters SA, Ashkenazi A, et al. (2020) Misfolded proteins bind and activate death receptor 5 to induce apoptosis during unresolved endoplasmic reticulum stress. Elife. 9 |
| Shemorry A, Harnoss JM, Guttman O, et al. (2019) Caspase-mediated cleavage of IRE1 controls apoptotic cell commitment during endoplasmic reticulum stress. Elife. 8 |
| Harnoss JM, Le Thomas A, Shemorry A, et al. (2019) Disruption of IRE1α through its kinase domain attenuates multiple myeloma. Proceedings of the National Academy of Sciences of the United States of America |
| Yang Y, Yeh SH, Madireddi S, et al. (2019) Tetravalent biepitopic targeting enables intrinsic antibody agonism of tumor necrosis factor receptor superfamily members. Mabs |
| Lam M, Marsters SA, Ashkenazi A, et al. (2019) Author response: Misfolded proteins bind and activate death receptor 5 to trigger apoptosis during unresolved endoplasmic reticulum stress Elife |
| Shemorry A, Harnoss JM, Guttman O, et al. (2019) Author response: Caspase-mediated cleavage of IRE1 controls apoptotic cell commitment during endoplasmic reticulum stress Elife |
| Chang TK, Lawrence DA, Lu M, et al. (2018) Coordination between Two Branches of the Unfolded Protein Response Determines Apoptotic Cell Fate. Molecular Cell. 71: 629-636.e5 |
| Lam M, Lawrence DA, Ashkenazi A, et al. (2018) Confirming a critical role for death receptor 5 and caspase-8 in apoptosis induction by endoplasmic reticulum stress. Cell Death and Differentiation |
| Ashkenazi A, Fairbrother WJ, Leverson JD, et al. (2017) From basic apoptosis discoveries to advanced selective BCL-2 family inhibitors. Nature Reviews. Drug Discovery |