Hongkang Zhang
Affiliations: | Neuroscience | Johns Hopkins University School of Medicine, Baltimore, MD, United States |
Area:
ion channelGoogle:
"Hongkang Zhang"Mean distance: 15.67 (cluster 11) | S | N | B | C | P |
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Publications
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| Zhang H, Werley K, Liu P, et al. (2020) An All-Optical Electrophysiology Screening Platform to Identify Nav Channel Modulators as Pain Therapeutics Biophysical Journal. 118: 325a |
| Zhang H, Reichert E, Cohen AE. (2016) Optical electrophysiology for probing function and pharmacology of voltage-gated ion channels. Elife. 5 |
| Zhang H, Reichert E, Cohen AE. (2016) Author response: Optical electrophysiology for probing function and pharmacology of voltage-gated ion channels Elife |
| Zhang H, Zou B, Du F, et al. (2015) Reporting sodium channel activity using calcium flux: pharmacological promiscuity of cardiac Nav1.5. Molecular Pharmacology. 87: 207-17 |
| Bruner JK, Zou B, Zhang H, et al. (2014) Identification of novel small molecule modulators of K2P18.1 two-pore potassium channel. European Journal of Pharmacology. 740: 603-10 |
| Fan L, Guan X, Wang W, et al. (2014) Impaired neuropathic pain and preserved acute pain in rats overexpressing voltage-gated potassium channel subunit Kv1.2 in primary afferent neurons. Molecular Pain. 10: 8 |
| Zhao X, Tang Z, Zhang H, et al. (2013) A long noncoding RNA contributes to neuropathic pain by silencing Kcna2 in primary afferent neurons. Nature Neuroscience. 16: 1024-31 |
| Yu H, Lin Z, Mattmann ME, et al. (2013) Dynamic subunit stoichiometry confers a progressive continuum of pharmacological sensitivity by KCNQ potassium channels. Proceedings of the National Academy of Sciences of the United States of America. 110: 8732-7 |
| Huang F, Zhang H, Wu M, et al. (2012) Calcium-activated chloride channel TMEM16A modulates mucin secretion and airway smooth muscle contraction. Proceedings of the National Academy of Sciences of the United States of America. 109: 16354-9 |
| Zhang H, Zou B, Yu H, et al. (2012) Modulation of hERG potassium channel gating normalizes action potential duration prolonged by dysfunctional KCNQ1 potassium channel. Proceedings of the National Academy of Sciences of the United States of America. 109: 11866-71 |