Jonathan Glass
Affiliations: | Neurology | Emory University, Atlanta, GA |
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Children
Sign in to add trainee| Lindsey R. Fischer | grad student | 2009 | Emory |
| Terrell E. Brotherton | grad student | 2012 | Emory |
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Publications
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| Pant DC, Parameswaran J, Rao L, et al. (2022) ALS-linked KIF5A ΔExon27 mutant causes neuronal toxicity through gain-of-function. Embo Reports. e54234 |
| Glass JD. (2020) Stathmin-2: adding another piece to the puzzle of TDP-43 proteinopathies and neurodegeneration. The Journal of Clinical Investigation |
| Miller T, Cudkowicz M, Shaw PJ, et al. (2020) Phase 1-2 Trial of Antisense Oligonucleotide Tofersen for ALS. The New England Journal of Medicine. 383: 109-119 |
| Wright ML, Fournier C, Houser MC, et al. (2018) Potential Role of the Gut Microbiome in ALS: A Systematic Review. Biological Research For Nursing. 1099800418784202 |
| Smith BN, Topp SD, Fallini C, et al. (2017) Mutations in the vesicular trafficking protein annexin A11 are associated with amyotrophic lateral sclerosis. Science Translational Medicine. 9 |
| Ayers JI, Diamond J, Sari A, et al. (2016) Distinct conformers of transmissible misfolded SOD1 distinguish human SOD1-FALS from other forms of familial and sporadic ALS. Acta Neuropathologica |
| Fischer-Hayes LR, Brotherton T, Glass JD. (2013) Axonal degeneration in the peripheral nervous system: implications for the pathogenesis of amyotrophic lateral sclerosis. Experimental Neurology. 246: 6-13 |
| Lee SM, Sha D, Mohammed AA, et al. (2013) Motor and sensory neuropathy due to myelin infolding and paranodal damage in a transgenic mouse model of Charcot-Marie-Tooth disease type 1C. Human Molecular Genetics. 22: 1755-70 |
| Brotherton TE, Li Y, Glass JD. (2013) Cellular toxicity of mutant SOD1 protein is linked to an easily soluble, non-aggregated form in vitro. Neurobiology of Disease. 49: 49-56 |
| Wu CH, Fallini C, Ticozzi N, et al. (2012) Mutations in the profilin 1 gene cause familial amyotrophic lateral sclerosis. Nature. 488: 499-503 |