Jonathan R. Roth
Affiliations: | University of Alabama, Birmingham, Birmingham, AL, United States |
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| Davis SE, Roth JR, Aljabi Q, et al. (2021) Delivering progranulin to neuronal lysosomes protects against excitotoxicity. The Journal of Biological Chemistry. 100993 |
| Voskobiynyk Y, Roth JR, Cochran JN, et al. (2020) Alzheimer's disease risk gene induces Tau-dependent network hyperexcitability. Elife. 9 |
| Voskobiynyk Y, Roth JR, Cochran JN, et al. (2020) Author response: Alzheimer’s disease risk gene BIN1 induces Tau-dependent network hyperexcitability Elife |
| Voskobiynyk Y, Roth JR, Cochran JN, et al. (2020) Tau‐dependent regulation of neuronal activity by the Alzheimer’s disease risk gene BIN1 Alzheimer's & Dementia. 16 |
| Arrant AE, Roth JR, Boyle NR, et al. (2019) Impaired β-glucocerebrosidase activity and processing in frontotemporal dementia due to progranulin mutations. Acta Neuropathologica Communications. 7: 218 |
| Rush T, Roth JR, Thompson SJ, et al. (2019) A peptide inhibitor of Tau-SH3 interactions ameliorates amyloid-β toxicity. Neurobiology of Disease. 104668 |
| Arrant AE, Boyle NR, Roth JR, et al. (2019) P1-186: IMPAIRED β-GLUCOCEREBROSIDASE ACTIVITY AND PROCESSING IN FRONTOTEMPORAL DEMENTIA DUE TO PROGRANULIN MUTATIONS Alzheimers & Dementia. 15 |
| Voskobiynyk Y, Cochran JN, Rush T, et al. (2006) P3-106: THE ALZHEIMER'S DISEASE RISK GENE BIN1 REGULATES NETWORK HYPEREXCITABILITY Alzheimer's & Dementia. 14: P1108-P1108 |