Elsa Quintana
Affiliations: | Associate Director | Revolution Medicines | |
| 2005-2011 | University of Michigan, Ann Arbor, Ann Arbor, MI |
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Publications
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| Quintana E, Schulze CJ, Myers DR, et al. (2020) Allosteric inhibition of SHP2 stimulates anti-tumor immunity by transforming the immunosuppressive environment. Cancer Research |
| Shifrin NT, Lee GJ, Omaque N, et al. (2020) Abstract 2837: Dual inhibition of SHP2 and CDK4/6 leads to immunological memory and immune-mediated anti-tumor activity in a mouse syngeneic model of breast cancer Tumor Biology |
| Quintana E, Schulze CJ, Choy TJ, et al. (2019) Abstract 5019: Allosteric inhibition of SHP2 suppresses CSF1R signaling and selectively reduces viability of M2 tumor associated macrophages contributing to anti-tumor immunity Cancer Research. 79: 5019-5019 |
| Eskiocak U, Ramesh V, Gill JG, et al. (2016) Erratum: Synergistic effects of ion transporter and MAP kinase pathway inhibitors in melanoma. Nature Communications. 7: 13080 |
| Eskiocak U, Ramesh V, Gill JG, et al. (2016) Synergistic effects of ion transporter and MAP kinase pathway inhibitors in melanoma. Nature Communications. 7: 12336 |
| Chartier C, Ma S, Antic D, et al. (2015) Abstract 2322: The Hippo signaling pathway mediates BMP inhibition of cancer stem cells Cancer Research. 75: 2322-2322 |
| Verhaegen ME, Mangelberger D, Weick JW, et al. (2014) Merkel cell carcinoma dependence on bcl-2 family members for survival. The Journal of Investigative Dermatology. 134: 2241-50 |
| Piskounova E, Quintana E, Shackleton M, et al. (2013) Abstract C9: Human melanoma metastasis in NSG mice correlates with clinical outcome in patients Cancer Research. 73 |
| Quintana E, Piskounova E, Shackleton M, et al. (2012) Human melanoma metastasis in NSG mice correlates with clinical outcome in patients. Science Translational Medicine. 4: 159ra149 |
| Joseph NM, He S, Quintana E, et al. (2011) Enteric glia are multipotent in culture but primarily form glia in the adult rodent gut. The Journal of Clinical Investigation. 121: 3398-411 |