Pier Giorgio Mastroberardino, PhD

Affiliations: 
Erasmus Medical Center Rotterdam, Rotterdam, Netherlands 
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"Pier Mastroberardino"
Mean distance: 17.21 (cluster 32)
 
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Publications

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D'Errico M, Parlanti E, Pascucci B, et al. (2021) The interplay between mitochondrial functionality and genome integrity in the prevention of human neurologic diseases. Archives of Biochemistry and Biophysics. 710: 108977
Milanese C, Gabriels S, Barnhoorn S, et al. (2020) Gender biased neuroprotective effect of Transferrin Receptor 2 deletion in multiple models of Parkinson's disease. Cell Death and Differentiation
Vandervore LV, Schot R, Milanese C, et al. (2019) TMX2 Is a Crucial Regulator of Cellular Redox State, and Its Dysfunction Causes Severe Brain Developmental Abnormalities. American Journal of Human Genetics
Milanese C, Bombardieri CR, Sepe S, et al. (2019) DNA damage and transcription stress cause ATP-mediated redesign of metabolism and potentiation of anti-oxidant buffering. Nature Communications. 10: 4887
Milanese C, Payán-Gómez C, Galvani M, et al. (2019) Peripheral mitochondrial function correlates with clinical severity in idiopathic Parkinson's disease. Movement Disorders : Official Journal of the Movement Disorder Society
Cerri S, Milanese C, Mastroberardino PG. (2019) Endocytic iron trafficking and mitochondria in Parkinson's disease. The International Journal of Biochemistry & Cell Biology. 110: 70-74
Milanese C, Payán-Gómez C, Mastroberardino PG. (2019) Cysteine oxidation and redox signaling in dopaminergic neurons physiology and in Parkinson’s disease Current Opinion in Physiology. 9: 73-78
Milanese C, Cerri S, Ulusoy A, et al. (2018) Activation of the DNA damage response in vivo in synucleinopathy models of Parkinson's disease. Cell Death & Disease. 9: 818
Milanese C, Tapias V, Gabriels S, et al. (2017) Mitochondrial complex I reversible S-nitrosation improves bioenergetics and is protective in Parkinson's disease. Antioxidants & Redox Signaling
Zambetti NA, Ping Z, Chen S, et al. (2016) Mesenchymal Inflammation Drives Genotoxic Stress in Hematopoietic Stem Cells and Predicts Disease Evolution in Human Pre-leukemia. Cell Stem Cell
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