Gang Zhang
Affiliations: | University of Nebraska Medical Center, Omaha, NE, United States |
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Parents
Sign in to add mentorHoward E. Gendelman | grad student | 2009-2014 | University of Nebraska Medical Center |
Stephen Spector | post-doc | 2014-2019 | UCSD |
Warner C. Greene | post-doc | 2019-2020 | (Microtree) |
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Publications
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Kong W, Frouard J, Xie G, et al. (2024) Neuroinflammation generated by HIV-infected microglia promotes dysfunction and death of neurons in human brain organoids. Pnas Nexus. 3: pgae179 |
Luo X, Frouard J, Zhang G, et al. (2022) Subsets of Tissue CD4 T Cells Display Different Susceptibilities to HIV Infection and Death: Analysis by CyTOF and Single Cell RNA-seq. Frontiers in Immunology. 13: 883420 |
Campbell GR, Zhuang J, Zhang G, et al. (2021) CD4+ T cell-mimicking nanoparticles encapsulating DIABLO/SMAC mimetics broadly neutralize HIV-1 and selectively kill HIV-1-infected cells. Theranostics. 11: 9009-9021 |
Zhang G, Huang X. (2021) Killing HIV-infected resting central memory CD4 T cells by targeting inhibitor of apoptosis proteins-inhibited autophagy. Antiviral Chemistry & Chemotherapy. 29: 2040206620980888 |
Zhang G, Campbell GR, Zhang Q, et al. (2020) CD4 T Cell-Mimicking Nanoparticles Broadly Neutralize HIV-1 and Suppress Viral Replication through Autophagy. Mbio. 11 |
Campbell GR, To RK, Zhang G, et al. (2020) SMAC mimetics induce autophagy-dependent apoptosis of HIV-1-infected macrophages. Cell Death & Disease. 11: 590 |
Zhang G, Luk BT, Wei X, et al. (2019) Selective cell death of latently HIV-infected CD4 T cells mediated by autosis inducing nanopeptides. Cell Death & Disease. 10: 419 |
Wei X, Zhang G, Ran D, et al. (2018) T-Cell-Mimicking Nanoparticles Can Neutralize HIV Infectivity. Advanced Materials (Deerfield Beach, Fla.). e1802233 |
Zhang G, Luk BT, Hamidy M, et al. (2018) Induction of a Na/K-ATPase-Dependent Form of Autophagy Triggers Preferential Cell Death of Human Immunodeficiency Virus Type-1-Infected Macrophages. Autophagy |
Kiyota T, Machhi J, Lu Y, et al. (2018) URMC-099 facilitates amyloid-β clearance in a murine model of Alzheimer's disease. Journal of Neuroinflammation. 15: 137 |