Teresa Rubio
Affiliations: | IDIBELL |
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| Viana R, Rubio T, Campos-Rodríguez Á, et al. (2024) Glial alterations in the glutamatergic and GABAergic signaling pathways in a mouse model of Lafora disease, a severe form of progressive myoclonus epilepsy. Biorxiv : the Preprint Server For Biology |
| Sanz P, Rubio T, Garcia-Gimeno MA. (2024) Neuroinflammation and Epilepsy: From Pathophysiology to Therapies Based on Repurposing Drugs. International Journal of Molecular Sciences. 25 |
| Rubio T, Campos-Rodríguez Á, Sanz P. (2023) Beneficial Effect of Fingolimod in a Lafora Disease Mouse Model by Preventing Reactive Astrogliosis-Derived Neuroinflammation and Brain Infiltration of T-lymphocytes. Molecular Neurobiology |
| Rubio T, Viana R, Moreno-Estellés M, et al. (2022) TNF and IL6/Jak2 signaling pathways are the main contributors of the glia-derived neuroinflammation present in Lafora disease, a fatal form of progressive myoclonus epilepsy. Neurobiology of Disease. 176: 105964 |
| Sanz P, Rubio T, Garcia-Gimeno MA. (2013) AMPKβ subunits: more than just a scaffold in the formation of AMPK complex. The Febs Journal. 280: 3723-33 |
| Rubio T, Vernia S, Sanz P. (2013) Sumoylation of AMPKβ2 subunit enhances AMP-activated protein kinase activity. Molecular Biology of the Cell. 24: 1801-11, S1-4 |
| Romá-Mateo C, Moreno D, Vernia S, et al. (2011) Lafora disease E3-ubiquitin ligase malin is related to TRIM32 at both the phylogenetic and functional level. Bmc Evolutionary Biology. 11: 225 |
| Vernia S, Rubio T, Heredia M, et al. (2009) Increased endoplasmic reticulum stress and decreased proteasomal function in lafora disease models lacking the phosphatase laforin. Plos One. 4: e5907 |
| Vernia S, Solaz-Fuster MC, Gimeno-Alcañiz JV, et al. (2009) AMP-activated protein kinase phosphorylates R5/PTG, the glycogen targeting subunit of the R5/PTG-protein phosphatase 1 holoenzyme, and accelerates its down-regulation by the laforin-malin complex. The Journal of Biological Chemistry. 284: 8247-55 |