Reisuke H. Takahashi, M.D., Ph.D.

Affiliations: 
Anatomic Pathology Tokyo Medical University, Los Angeles, CA, United States 
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"Reisuke Takahashi"
Bio:

associate professor

Mean distance: 15.66 (cluster 28)
 
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Publications

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Takahashi RH, Yokotsuka M, Tobiume M, et al. (2021) Accumulation of cellular prion protein within β-amyloid oligomer plaques in aged human brains. Brain Pathology (Zurich, Switzerland). e12941
Willén K, Sroka A, Takahashi RH, et al. (2017) Heterogeneous Association of Alzheimer's Disease-Linked Amyloid-β and Amyloid-β Protein Precursor with Synapses. Journal of Alzheimer's Disease : Jad
Takahashi RH, Nagao T, Gouras GK. (2017) Plaque formation and the intraneuronal accumulation of β-amyloid in Alzheimer's disease. Pathology International
Takahashi RH, Capetillo-Zarate E, Lin MT, et al. (2013) Accumulation of intraneuronal β-amyloid 42 peptides is associated with early changes in microtubule-associated protein 2 in neurites and synapses. Plos One. 8: e51965
Takahashi RH, Tobiume M, Sato Y, et al. (2011) Accumulation of cellular prion protein within dystrophic neurites of amyloid plaques in the Alzheimer's disease brain. Neuropathology : Official Journal of the Japanese Society of Neuropathology. 31: 208-14
Gouras GK, Tampellini D, Takahashi RH, et al. (2010) Intraneuronal beta-amyloid accumulation and synapse pathology in Alzheimer's disease. Acta Neuropathologica. 119: 523-41
Takahashi RH, Capetillo-Zarate E, Lin MT, et al. (2010) Co-occurrence of Alzheimer's disease ß-amyloid and τ pathologies at synapses. Neurobiology of Aging. 31: 1145-52
Capetillo-Zarate E, Gracia L, Takahashi RH, et al. (2010) O4-06-02: High resolution 3D volumetric visualization of the progression of intraneuronal Aβ and plaque pathology in brain Alzheimer's & Dementia. 6: S159-S160
Capetillo-Zarate E, Takahashi RH, Almeida CG, et al. (2008) P4-260: Intraneuronal Aβ42 and microtubule-associated protein pathologies begin in synapses Alzheimer's & Dementia. 4: T747-T748
Tampellini D, Magrané J, Takahashi RH, et al. (2007) Internalized antibodies to the Abeta domain of APP reduce neuronal Abeta and protect against synaptic alterations. The Journal of Biological Chemistry. 282: 18895-906
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