We are testing a new system for linking grants to scientists.
The funding information displayed below comes from the
NIH Research Portfolio Online Reporting Tools and the
NSF Award Database.
The grant data on this page is limited to grants awarded in the United States and is thus partial. It can nonetheless be used to understand how funding patterns influence mentorship networks and vice-versa, which has deep implications on how research is done.
You can help! If you notice any innacuracies, please
sign in and mark grants as correct or incorrect matches.
Sign in to see low-probability grants and correct any errors in linkage between grants and researchers.
High-probability grants
According to our matching algorithm, Isadora F. Bielsky is the likely recipient of the following grants.
Years |
Recipients |
Code |
Title / Keywords |
Matching score |
2003 — 2004 |
Bielsky, Isadora F |
F31Activity Code Description: To provide predoctoral individuals with supervised research training in specified health and health-related areas leading toward the research degree (e.g., Ph.D.). |
Vasopressin Receptors, Social Behaviors, &Anxiety
[unreadable] DESCRIPTION (provided by applicant): Vasopressin (AVP) is a peptide produced in the brain and released both centrally and peripherally. The central actions of AVP include effects on both social and non-social behaviors. Two main subtypes of AVP receptors exist in the brain, namely V1alpha and V1beta. It is unclear via which receptor subtype AVP acts to affect these behaviors. Understanding the mechanisms behind AVP's effects on anxiety and social recognition is particularly important given the relationship between these behaviors and human disorders such as anxiety disorder and autism. The aims of this proposal are to use mice with a null mutation in the V1alpha receptor (V1aRKO) to better understand how AVP affects anxiety and social behavior. In the first aim, the V1aRKO mice will be behaviorally and neurochemically phenotyped to assess the effects of a null mutation in the V1aR. In the second aim, differences in c-fos-IR between the V1aRKOs and WT animals will be used to determine neuroanatomical regions involved in anxiety and social behavior. In the third aim, a V1aR viral vector will be used to re-express the V1aR in specific neuroanatomical regions, in an attempt to alter the anxiety and social behavior of the KO mice. These experiments will further our understanding of the mechanisms involved in AVP mediated anxiety and social behavior. [unreadable] [unreadable]
|
1 |