Area:
Nutrition, Cognitive Psychology, Behavioral Psychology
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High-probability grants
According to our matching algorithm, Kevin Laugero is the likely recipient of the following grants.
| Years |
Recipients |
Code |
Title / Keywords |
Matching
score |
| 2001 — 2002 |
Laugero, Kevin D |
F32Activity Code Description:
To provide postdoctoral research training to individuals to broaden their scientific background and extend their potential for research in specified health-related areas. |
Metabolic Regulation of Basal Activity in the Hpa Axis @ University of California San Francisco
DESCRIPTION: (Scanned from the applicant's abstract) Glucocorticoids are assumed to act at the brain to exert their feedback effects. However, there is little, if any evidence that glucocorticoid feedback acts at specific brain sites to maintain basal CRF and ACTH secretion. There is a major effect of glucocorticoids on energy balance exerted in the periphery (on liver, fat, muscle). We hypthesize that glucocorticoid feedback on basal CRF and ACTH secretion is not on brain, but is indirect and consequence of systemic actions of glucocorticoids on metabolism, stimulating metabolic signals that in turn act on brain to normalize activity in the HPA axis (i.e., a brain- glucocorticoid-metabolic-brain axis). We have a powerful tool ( the use of sucrose) with which to test this hypothesis. Adrenalectomized rats drinking sucrose normalize both their behaviors and energy balance compared to Sham. Sucrose also normalizes CRF mRNA in the paraventricular nuclei and amygdala and dopamine-beta-Hydroxylase (DBH) mRNA in catecholaminergic cell groups (nucleus tractus solitarius and locus coeruleus) in ADX rats. We propose to test our hypothesis by determining whether 1. sucrose-induced changes in DBH and CRF are mediated from brainstem to hypothalamus, vice-versa or independently, 2. sucrose-induced changes in paraventricular nuclei (PVN) CRF are dependent on the bed mucleus of the stria terminalis (BNST) or amygdala, and 3. the metabolic signals of sucrose drinking affect the brain via: (a) neural afferents from hepathic vagus, (b) leptin, and (c) brain malony-CoA system. Indiviuals suffering from depression, anorexia, type II diabetes, or Abdominal obesity usually present with altered activity in the HPA axis. These studies will be important for developing a new framework with which to understand the etiologies of such health problems.
|
0.941 |
| 2008 — 2009 |
Laugero, Kevin D |
P51Activity Code Description:
To support centers which include a multidisciplinary and multi-categorical core research program using primate animals and to maintain a large and varied primate colony which is available to affiliated, collaborative, and visiting investigators for basic and applied biomedical research and training. |
Metabolomic &Hypothalamic-Pituitary-Adrenal Responses to Chronic Social Stress @ University of California Davis
ADRGND; Address; Adrenal Glands; Adrenals; Animal Model; Animal Models and Related Studies; CRISP; Chronic; Chronic stress; Computer Retrieval of Information on Scientific Projects Database; Funding; Grant; Hypophysis; Hypophysis Cerebri; Hypothalamic structure; Hypothalamus; Institution; Investigators; Metabolic; Modeling; NIH; National Institutes of Health; National Institutes of Health (U.S.); Nervous System, Pituitary; Neuroendocrine; Neuroendocrine System; Neurosecretory Systems; Pituitary; Pituitary Gland; Psychosocial Stress; Research; Research Personnel; Research Resources; Researchers; Resources; Source; United States National Institutes of Health; hypothalamic; measurement of metabolism; metabolomics; model organism; response; social stress; suprarenal gland
|
0.958 |