Shinghua Ding
Affiliations: | 2008- | Bioengineering | University of Missouri - Columbia, Columbia, MO, United States |
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"Shinghua Ding"Mean distance: 17.39 (cluster 6)
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Sign in to add traineeYicheng Xie | grad student | 2008-2011 | University of Missouri - Columbia |
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Publications
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Cegielski V, Chakrabarty R, Ding S, et al. (2022) Acid-Sensing Ion Channels in Glial Cells. Membranes. 12 |
Zhang N, Zhang Z, Ozden I, et al. (2022) Imaging Mitochondrial Ca2+ Uptake in Astrocytes and Neurons using Genetically Encoded Ca2+ Indicators (GECIs). Journal of Visualized Experiments : Jove |
Lundt S, Ding S. (2021) Non-cell autonomous effect of neuronal nicotinamide phosphoribosyl transferase on the function of neuromuscular junctions. Neural Regeneration Research. 16: 302-303 |
Zhang N, Zhang Z, He R, et al. (2020) GLAST-CreER mediated deletion of GDNF increases brain damage and exacerbates long-term stroke outcomes after focal ischemic stroke in mouse model. Glia |
Lundt S, Zhang N, Wang X, et al. (2020) The effect of NAMPT deletion in projection neurons on the function and structure of neuromuscular junction (NMJ) in mice. Scientific Reports. 10: 99 |
Wang X, Zhang Z, Zhang N, et al. (2019) Subcellular NAMPT-mediated NAD salvage pathways and their roles in bioenergetics and neuronal protection after ischemic injury. Journal of Neurochemistry |
Wang X, Zhang Q, Bao R, et al. (2017) Deletion of Nampt in Projection Neurons of Adult Mice Leads to Motor Dysfunction, Neurodegeneration, and Death. Cell Reports. 20: 2184-2200 |
Wang X, Ding S. (2016) Pre-B-cell colony-enhancing factor as a target for protecting against apoptotic neuronal death and mitochondrial damage in ischemia. Neural Regeneration Research. 11: 1914-1915 |
Wang X, Li H, Ding S. (2016) Pre-B-cell colony-enhancing factor protects against apoptotic neuronal death and mitochondrial damage in ischemia. Scientific Reports. 6: 32416 |
Li H, Xie Y, Zhang N, et al. (2015) Disruption of IP3R2-mediated Ca(2+) signaling pathway in astrocytes ameliorates neuronal death and brain damage while reducing behavioral deficits after focal ischemic stroke. Cell Calcium |