Brooks Pond
Affiliations: | 2004-2006 | Pharmacy | Eastern Tennessee State School of Pharmacy |
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"Brooks Pond"Mean distance: 18.13 (cluster 32) | S | N | B | C | P |
Parents
Sign in to add mentorRichard Jay Smeyne | post-doc | 2004-2006 | Eastern Tennessee State School of Pharmacy |
Richard Jay Smeyne | post-doc | 2004-2006 | Eastern Tennessee State School of Pharmacy |
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Publications
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Oakes HV, McWethy D, Ketchem S, et al. (2021) Effect of Chronic Methylphenidate Treatment in a Female Experimental Model of Parkinsonism. Neurotoxicity Research |
Oakes HV, Ketchem S, Hall AN, et al. (2019) Chronic methylphenidate induces increased quinone production and subsequent depletion of the antioxidant glutathione in the striatum. Pharmacological Reports : Pr. 71: 1289-1292 |
Oakes HV, Ketchem S, Hall AN, et al. (2019) Chronic methylphenidate induces increased quinone production and subsequent depletion of the antioxidant glutathione in the striatum. Pharmacological Reports : Pr. 71: 1289-1292 |
Sadasivan S, Pond BB, Pani AK, et al. (2012) Methylphenidate exposure induces dopamine neuron loss and activation of microglia in the basal ganglia of mice. Plos One. 7: e33693 |
Sadasivan S, Pond BB, Pani AK, et al. (2012) Correction: Methylphenidate Exposure Induces Dopamine Neuron Loss and Activation of Microglia in the Basal Ganglia of Mice Plos One. 7 |
Smeyne M, Boyd J, Shepherd KR, et al. (2007) GSTπ expression mediates dopaminergic neuron sensitivity in experimental parkinsonism Proceedings of the National Academy of Sciences of the United States of America. 104: 1977-1982 |