Harinder Singh, Ph.D.
Affiliations: | 2014- | Physiology and Biophysics | University of Illinois at Chicago, Chicago, IL, United States |
Area:
NeuroscienceWebsite:
www.grewalsynapse.comGoogle:
"Harinder Singh"Bio:
https://scholar.google.com/citations?user=Sgr0uKoAAAAJ&hl=en
Cell biologist with years of working experience in academia and industry. Studied several different proteins and mechanisms important in human patho-physiology employing wide variety of biochemical methods. My research so far has been cross-disciplinary by employing concepts learned from the field of cardiovascular biology into the field of neuroscience in understanding molecular basis of debilitating psychiatric diseases.
Mean distance: 25.28 (cluster 52)
Parents
Sign in to add mentorPrabeer K. Sinha | research assistant | 1997-2000 | Delhi University, India | |
KS Krishnan | research assistant | 2000-2001 | Tata Institute of Fundamental Research, Mumbai, India | |
Mitradas Madhav Panicker | research assistant | 2000-2001 | National Center for Biological Science | |
K Muralidhar | research assistant | 2000-2002 | Delhi University, India | |
BN Mallick | research assistant | 2004-2005 | Jawaharlal Nehru University, New Delhi | |
Judy M. Teale | research assistant | 2005-2007 | UT San Antonio | |
Victor Rizzo | grad student | 2009-2014 | Temple University | |
(Caveolae as spatio-temporal compartments for ROS/RNS generation and nitroxidative stress signaling.) | ||||
Mark M. Rasenick | post-doc | 2014- | College of Medicine |
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Publications
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Bertels Z, Singh H, Dripps I, et al. (2021) Neuronal complexity is attenuated in preclinical models of migraine and restored by HDAC6 inhibition. Elife. 10 |
Singh H, Chmura J, Bhaumik R, et al. (2020) Membrane-associated α-tubulin is less acetylated in postmortem prefrontal cortex from depressed subjects relative to controls: cytoskeletal dynamics, HDAC6 and depression. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience |
Sural-Fehr T, Singh H, Cantuti-Catelvetri L, et al. (2019) Inhibition of IGF-1-PI3K-Akt-mTORC2 in lipid rafts increases neuronal vulnerability in a genetic lysosomal glycosphingolipidosis. Disease Models & Mechanisms |
Singh H, Wray N, Schappi JM, et al. (2019) Correction: Disruption of lipid-raft localized Gαs/tubulin complexes by antidepressants: a unique feature of HDAC6 inhibitors, SSRI, and tricyclic compounds. Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology |
Wray NH, Schappi JM, Singh H, et al. (2018) NMDAR-independent, cAMP-dependent antidepressant actions of ketamine. Molecular Psychiatry |
Singh H, Wray N, Schappi JM, et al. (2018) Disruption of lipid-raft localized Gα/tubulin complexes by antidepressants: a unique feature of HDAC6 inhibitors, SSRI and tricyclic compounds. Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology |
Wray N, Schappi J, Singh H, et al. (2017) 793. Camp-Dependent and NMDAR-Independent Action of Ketamine in a Cellular Model System Biological Psychiatry. 81: S322 |
Woitek F, Zentilin L, Hoffman NE, et al. (2015) Intracoronary Cytoprotective Gene Therapy: A Study of VEGF-B167 in a Pre-Clinical Animal Model of Dilated Cardiomyopathy. Journal of the American College of Cardiology. 66: 139-53 |
Gabunia K, Ellison SP, Singh H, et al. (2012) Interleukin-19 (IL-19) induces heme oxygenase-1 (HO-1) expression and decreases reactive oxygen species in human vascular smooth muscle cells. The Journal of Biological Chemistry. 287: 2477-84 |
Verma S, Trivedi JK, Singh H, et al. (1999) Serum lipid profile in suicide attempters. Indian Journal of Psychiatry. 41: 300-6 |