Tyler K. Best

Affiliations: 
Northwestern University, Evanston, IL 
Area:
Dendritic integration
Google:
"Tyler Best"
Mean distance: 14.42 (cluster 6)
 
SNBCP
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Publications

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Best TK, Marson L, Thor KB, et al. (2013) Synaptic activation of bulbospongiosus motoneurons via dorsal gray commissural inputs. Journal of Neurophysiology. 109: 58-67
Best TK, Cramer NP, Chakrabarti L, et al. (2012) Dysfunctional hippocampal inhibition in the Ts65Dn mouse model of Down syndrome. Experimental Neurology. 233: 749-57
Sheffield ME, Best TK, Mensh BD, et al. (2011) Slow integration leads to persistent action potential firing in distal axons of coupled interneurons. Nature Neuroscience. 14: 200-7
Cramer NP, Best TK, Stoffel M, et al. (2010) GABAB-GIRK2-mediated signaling in Down syndrome. Advances in Pharmacology (San Diego, Calif.). 58: 397-426
Chakrabarti L, Best TK, Cramer NP, et al. (2010) Olig1 and Olig2 triplication causes developmental brain defects in Down syndrome. Nature Neuroscience. 13: 927-34
Best TK, Cho-Clark M, Siarey RJ, et al. (2008) Speeding of miniature excitatory post-synaptic currents in Ts65Dn cultured hippocampal neurons. Neuroscience Letters. 438: 356-61
Best TK, Siarey RJ, Galdzicki Z. (2007) Ts65Dn, a mouse model of Down syndrome, exhibits increased GABAB-induced potassium current. Journal of Neurophysiology. 97: 892-900
Siarey RJ, Kline-Burgess A, Cho M, et al. (2006) Altered signaling pathways underlying abnormal hippocampal synaptic plasticity in the Ts65Dn mouse model of Down syndrome. Journal of Neurochemistry. 98: 1266-77
Harashima C, Jacobowitz DM, Witta J, et al. (2006) Abnormal expression of the G-protein-activated inwardly rectifying potassium channel 2 (GIRK2) in hippocampus, frontal cortex, and substantia nigra of Ts65Dn mouse: a model of Down syndrome. The Journal of Comparative Neurology. 494: 815-33
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