Joseph J. Belanto, Ph.D.
Affiliations: | Molecular, Cellular, Developmental Biology and Genetics | University of Minnesota, Twin Cities, Minneapolis, MN |
Area:
Dystrophen-glycoprotein complexGoogle:
"Joseph Belanto"Mean distance: 19.53 (cluster 11)
Parents
Sign in to add mentorJames M. Ervasti | grad student | 2013 | UMN | |
(A Biochemical and Molecular Analysis of Functional Differences between Dystrophin and Utrophin.) |
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Publications
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Randazzo D, Khalique U, Belanto JJ, et al. (2019) Persistent upregulation of the β-tubulin tubb6, linked to muscle regeneration, is a source of microtubule disorganization in dystrophic muscle. Human Molecular Genetics |
Randazzo D, Khalique U, Belanto JJ, et al. (2018) Persistent upregulation of the β-tubulin tubb6, linked to muscle regeneration, is a source of microtubule disorganization in dystrophic muscle. Human Molecular Genetics |
Olthoff JT, Lindsay A, Abo-Zahrah R, et al. (2018) Loss of peroxiredoxin-2 exacerbates eccentric contraction-induced force loss in dystrophin-deficient muscle. Nature Communications. 9: 5104 |
McCourt JL, Talsness DM, Lindsay A, et al. (2017) Mouse models of two missense mutations in actin binding domain 1 of dystrophin associated with Duchenne or Becker muscular dystrophy. Human Molecular Genetics |
Cermak T, Curtin SJ, Gil-Humanes J, et al. (2017) A multi-purpose toolkit to enable advanced genome engineering in plants. The Plant Cell |
Belanto JJ, Olthoff JT, Mader TL, et al. (2016) Independent variability of microtubule perturbations associated with dystrophinopathy. Human Molecular Genetics. 25: 4951-4961 |
Belanto JJ, Olthoff JT, Mader TL, et al. (2016) Independent variability of microtubule perturbations associated with dystrophinopathy. Human Molecular Genetics |
Olthoff JT, Belanto JJ, Lindsay A, et al. (2016) Peroxiredoxin II Deficiency Contributes to Contraction-Induced Force Loss in the mdx Model of Duchenne Muscular Dystrophy Free Radical Biology and Medicine. 100: S107 |
Filareto A, Rinaldi F, Arpke RW, et al. (2015) Pax3-induced expansion enables the genetic correction of dystrophic satellite cells. Skeletal Muscle. 5: 36 |
Talsness DM, Belanto JJ, Ervasti JM. (2015) Disease-proportional proteasomal degradation of missense dystrophins. Proceedings of the National Academy of Sciences of the United States of America |