Bryce Vissel

Affiliations: 
2003-2008 Garvan Institute of Medical Research, Darlinghurst, New South Wales, Australia 
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"Bryce Vissel"
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Konen LM, Wright AL, Royle GA, et al. (2020) A new mouse line with reduced GluA2 Q/R site RNA editing exhibits loss of dendritic spines, hippocampal CA1-neuron loss, learning and memory impairments and NMDA receptor-independent seizure vulnerability. Molecular Brain. 13: 27
Stayte S, Laloli KJ, Rentsch P, et al. (2019) The kainate receptor antagonist UBP310 but not single deletion of GluK1, GluK2, or GluK3 subunits, inhibits MPTP-induced degeneration in the mouse midbrain. Experimental Neurology. 113062
Rentsch P, Stayte S, Morris GP, et al. (2019) Time dependent degeneration of the nigrostriatal tract in mice with 6-OHDA lesioned medial forebrain bundle and the effect of activin A on L-Dopa induced dyskinesia. Bmc Neuroscience. 20: 5
Clark IA, Vissel B. (2018) Therapeutic implications of how TNF links APOE, P-tau, α-synuclein, β-amyloid, and insulin resistance in neurodegenerative diseases. British Journal of Pharmacology
Clark IA, Vissel B. (2017) The meteorology of cytokine storms, and the clinical usefulness of this knowledge. Seminars in Immunopathology
Stayte S, Rentsch P, Tröscher AR, et al. (2017) Activin A Inhibits MPTP and LPS-Induced Increases in Inflammatory Cell Populations and Loss of Dopamine Neurons in the Mouse Midbrain In Vivo. Plos One. 12: e0167211
Clark IA, Vissel B. (2016) Excess cerebral TNF causing glutamate excitotoxicity rationalizes treatment of neurodegenerative diseases and neurogenic pain by anti-TNF agents. Journal of Neuroinflammation. 13: 236
Clark IA, Vissel B. (2015) A Neurologist's Guide to TNF Biology and to the Principles behind the Therapeutic Removal of Excess TNF in Disease. Neural Plasticity. 2015: 358263
Stayte S, Rentsch P, Li KM, et al. (2015) Activin A protects midbrain neurons in the 6-hydroxydopamine mouse model of Parkinson's disease. Plos One. 10: e0124325
Stayte S, Vissel B. (2014) Advances in non-dopaminergic treatments for Parkinson's disease. Frontiers in Neuroscience. 8: 113
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