Stanley C. Froehner

Affiliations: 
University of Washington, Seattle, Seattle, WA 
Area:
synapse formation, neuromuscular synapse
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"Stanley Froehner"
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Publications

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Kim MJ, Bible KL, Regnier M, et al. (2019) Simvastatin provides long-term improvement of left ventricular function and prevents cardiac fibrosis in muscular dystrophy. Physiological Reports. 7: e14018
Kim MJ, Whitehead NP, Bible KL, et al. (2018) Mice lacking α-,β1- and β2-syntrophins exhibit diminished function and reduced dystrophin expression in both cardiac and skeletal muscle. Human Molecular Genetics
Adams ME, Odom GL, Kim MJ, et al. (2018) Syntrophin binds directly to multiple spectrin-like repeats in dystrophin and mediates binding of nNOS to repeats 16-17. Human Molecular Genetics
Whitehead NP, Bible KL, Kim MJ, et al. (2016) Validation of ultrasonography for non-invasive assessment of diaphragm function in muscular dystrophy. The Journal of Physiology
Nanou E, Yan J, Whitehead NP, et al. (2016) Altered short-term synaptic plasticity and reduced muscle strength in mice with impaired regulation of presynaptic CaV2.1 Ca2+ channels. Proceedings of the National Academy of Sciences of the United States of America
Allen DG, Whitehead NP, Froehner SC. (2016) Absence of Dystrophin Disrupts Skeletal Muscle Signaling: Roles of Ca2+, Reactive Oxygen Species, and Nitric Oxide in the Development of Muscular Dystrophy. Physiological Reviews. 96: 253-305
Rebolledo DL, Kim MJ, Whitehead NP, et al. (2015) Sarcolemmal targeting of nNOSμ improves contractile function of mdx muscle. Human Molecular Genetics
Whitehead NP, Kim MJ, Bible KL, et al. (2015) A new therapeutic effect of simvastatin revealed by functional improvement in muscular dystrophy. Proceedings of the National Academy of Sciences of the United States of America
Eisinger K, Froehner SC, Adams ME, et al. (2015) Evaluation of the specificity of four commercially available antibodies to alpha-syntrophin. Analytical Biochemistry. 484: 99-101
Hebel T, Eisinger K, Neumeier M, et al. (2015) Lipid abnormalities in alpha/beta2-syntrophin null mice are independent from ABCA1. Biochimica Et Biophysica Acta. 1851: 527-36
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