Vishnu A. Cuddapah, Ph.D.
Affiliations: | Neurobiology | University of Alabama, Birmingham, Birmingham, AL, United States |
Area:
The Role of Neuroglia in Brain Function & DiseaseGoogle:
"Vishnu Cuddapah"Mean distance: 17.99 (cluster 11)
Parents
Sign in to add mentorHarald W. Sontheimer | grad student | 2012 | UAB | |
(Regulation of ClC-3 in human malignant glioma.) |
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Publications
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Kahanovitch U, Cuddapah VA, Pacheco NL, et al. (2018) MeCP2 Deficiency Leads to Loss of Glial Kir4.1. Eneuro. 5 |
Nwaobi SE, Cuddapah VA, Patterson KC, et al. (2016) The role of glial-specific Kir4.1 in normal and pathological states of the CNS. Acta Neuropathologica |
Cuddapah VA, Sinifunanya EN, Percy AK, et al. (2015) MeCP2 in the regulation of neural activity: Rett syndrome pathophysiological perspectives. Degenerative Neurological and Neuromuscular Disease. 5: 103-116 |
Campbell SL, Robel S, Cuddapah VA, et al. (2015) GABAergic disinhibition and impaired KCC2 cotransporter activity underlie tumor-associated epilepsy. Glia. 63: 23-36 |
Cuddapah VA, Robel S, Watkins S, et al. (2014) A neurocentric perspective on glioma invasion. Nature Reviews. Neuroscience. 15: 455-65 |
Cuddapah VA, Pillai RB, Shekar KV, et al. (2014) Methyl-CpG-binding protein 2 (MECP2) mutation type is associated with disease severity in Rett syndrome. Journal of Medical Genetics. 51: 152-8 |
Cuddapah VA, Turner KL, Seifert S, et al. (2013) Bradykinin-induced chemotaxis of human gliomas requires the activation of KCa3.1 and ClC-3. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 33: 1427-40 |
Cuddapah VA, Turner KL, Sontheimer H. (2013) Calcium entry via TRPC1 channels activates chloride currents in human glioma cells. Cell Calcium. 53: 187-94 |
McFerrin MB, Turner KL, Cuddapah VA, et al. (2012) Differential role of IK and BK potassium channels as mediators of intrinsic and extrinsic apoptotic cell death. American Journal of Physiology. Cell Physiology. 303: C1070-8 |
Cuddapah VA, Habela CW, Watkins S, et al. (2012) Kinase activation of ClC-3 accelerates cytoplasmic condensation during mitotic cell rounding. American Journal of Physiology. Cell Physiology. 302: C527-38 |