Asa Hatami, Ph.D.

Affiliations: 
Biological Sciences - Ph.D. University of California, Irvine, Irvine, CA 
Area:
Fertilization, Alzheimer disease
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"Asa Hatami"
Mean distance: 19.35 (cluster 28)
 
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Charles Glabe grad student 2013 UC Irvine
 (Conformation-specific Antibodies Distinguish Amyloid Polymorphisms Associated with Alzheimer's Disease.)
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Publications

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Foley AR, Finn TS, Kung T, et al. (2019) Trapping and Characterization of Non-Toxic Aβ42 Aggregation Intermediates. Acs Chemical Neuroscience
Bilousova T, Melnik M, Miyoshi E, et al. (2019) Apolipoprotein E/Aβ Complex Accumulates in AD Cortical Synapses via apoE Receptors and Is Enhanced by APOE4. The American Journal of Pathology
Bilousova T, Elias CJ, Melnik M, et al. (2019) P4-057: PURIFICATION AND CHARACTERIZATION OF SUBPOPULATIONS OF EXTRACELLULAR VESICLES FROM AD AND PD BRAIN AND THEIR SUPPRESSION BY NSMASE2 INHIBITORS Alzheimer's & Dementia. 15: P1294-P1295
Campagna J, Spilman P, Jagodzinska B, et al. (2018) A small molecule ApoE4-targeted therapeutic candidate that normalizes sirtuin 1 levels and improves cognition in an Alzheimer's disease mouse model. Scientific Reports. 8: 17574
Hatami A, Monjazeb S, Milton S, et al. (2017) Familial Alzheimer's disease mutations within the amyloid precursor protein alter the aggregation and conformation of the amyloid beta peptide. The Journal of Biological Chemistry
Knight EM, Kim SH, Kottwitz JC, et al. (2016) Effective anti-Alzheimer Aβ therapy involves depletion of specific Aβ oligomer subtypes. Neurology® Neuroimmunology & Neuroinflammation. 3: e237
Hatami A, Monjazeb S, Glabe C. (2015) The Anti-Amyloid-β Monoclonal Antibody 4G8 Recognizes a Generic Sequence-Independent Epitope Associated with α-Synuclein and Islet Amyloid Polypeptide Amyloid Fibrils. Journal of Alzheimer's Disease : Jad
Tan Z, Dai W, van Erp TG, et al. (2015) Huntington's disease cerebrospinal fluid seeds aggregation of mutant huntingtin. Molecular Psychiatry
Hatami A, Chesselet MF. (2015) Transgenic rodent models to study alpha-synuclein pathogenesis, with a focus on cognitive deficits. Current Topics in Behavioral Neurosciences. 22: 303-30
Hatami A, Albay R, Monjazeb S, et al. (2014) Monoclonal antibodies against Aβ42 fibrils distinguish multiple aggregation state polymorphisms in vitro and in Alzheimer disease brain. The Journal of Biological Chemistry. 289: 32131-43
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